1[1]Boyko OB, Burger PC, Shelburne JD, et al. Non - heme mechanisms for T1 shortening: pathology, CT, and MR elucidation. AJNR Am J Neuroradiol, 1992, 13: 1439- 1445.
2[2]Hayashi Y, Tachibana O, Muramatsu N, et al. Rathke cleft cyst: MR and biomedical analysis of cyst content. J Comput Assist Tomogr, 1999,23: 34 - 38.
3[3]Dell LA, Brown MS, Orrison WW, et al. Physiologic intracranial calcification with hyperintensity on MR imaging: case report and experimental model. AJNR Am J Neuroradiol, 1988, 9: 1145- 1148.
5[5]Fujioka M, Taoka T, Hiramatsu KI, et al. Delayed ischemic hyperintensity on T1 - weighted MRI in the caudoputamen cerebral cortex of humans after spectacular shrinking deficit. Stroke, 1999, 30: 1038-1042.
6[6]Fujioka M, Taoka T, Mastsuo Y, et al. Brief ischemic cerebral infarction changes. Stroke, 1999, 30: 1042- 1046.
7[7]Komiyama M, Nakajima H, Nishikawa M, et al. Serial MR observation of cortical laminar necrosis caused by brain infarction. Neuroradiology,1998, 40:771 - 777.
8[8]Komiyama M, Nishikawa M, Yasui T, et al. Cortical laminar necrosis in brain infarcts: chronological changes on MRI. Neuroradiology, 1997,39: 474 - 479.
9[9]Johansson L, Jansson JH, Boman K, et al. Tissue plasminogen activator, plasminogen activator inhibitor - 1, and tissue plasminogen activator/ plasminogen activator inhibitor- 1 complex as risk factors for the development of a first stroke. Stroke, 2000, 31:26- 32.
10[10]Suarez JI, Sunshine JL, Tart R, et al. Predictors of clinical improvement, angiographic recanalization, and intracranial hemorrhage after intra - arterial thrombolysis for acute ischemic stroke. Stroke, 1999,30:2094 - 2100.