1Zuchelli P, Santom A. Dialysis-induced hypotension: A fresh look at pathophysiology. Blood Purif, 1993, 11 : 5 -98.
2Beasley D, Brenner BM. Role of nitric oxide in hemodialysishypotension. Kidney Int, 1992,42(Supple 38) :96 - 100.
3Nishimura M, Takahashi H,Maruyama K, et al. Enhanced production of nitric oxide may be involved in acute hypotension during maintenance hemodialysis. Am J Kidney Dis,1998,31(5): 809-817.
4Yokokawa K, Mankus R, Saklayen MG, et al. Increased nitric oxide production in patients with hypotension during hemodialysis. Ann Intern Med, 1995,123(1) :35 - 37.
5Kang FS, Tevlin MT, Wang YB, et al. Hemodialysis hypotension: Interaction inhibitors, iNOS and the interdialytic period. Am J Med Sci,1999,317(1):9-21.
6Perazella MA. Niric oxide and hemodialysis-induced hypotension. Ann Intern Med, 1996,124(4) :454.
7Iwashina M, Hiram Y, Imai T, et al. Molecular cloning of endothelial, inducible NO synthase gene from rat aotic endothelial cell. Eur J Biochern, 1996, 237:668 - 673.
8Singh K, Balligand K, Fisher TA, et al. Glueoeortieoids increased osteopontin expression in cardiac myocytes and rnicrovascular endothelial cells. Role in regulation of iNOS. J Biol Chern, 1995,270: 28471 - 28478.
10Dinarello CA. lnterleukin-1 and tumor necrosis faeters and their naturally occurring antagonists during hemoclialysis.Kidney lnt, 1992, (Supple 38) : 68 - 77.