期刊文献+

短蛋白聚糖在脑损伤后表达上调的研究进展

Research progress in expression increase of brevican following brain injury
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作者 武弋 雷鹏
出处 《中华创伤杂志》 CAS CSCD 北大核心 2003年第11期697-698,共2页 Chinese Journal of Trauma
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参考文献13

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  • 2韩晞,董艳,卢亦成.短蛋白聚糖研究进展[J].生理科学进展,2002,33(3):248-250. 被引量:1
  • 3滕继平,张治宇,杨长伟,陈哲宇.硫酸软骨素蛋白聚糖在脑发育中的作用[J].生命的化学,2001,21(4):293-295. 被引量:4
  • 4Yamada H, Watanabe K, Shimonaka M, et al. Molecular cloning of brevican, a novelbrain proteoglycan of the aggrecan/versican family. J Biol Chem, 1994, 269:10119-10126.
  • 5Wu RR, Couchman JR, cDNA cloning of the basement membrane chondroitin sulfate proteoglycan core protein, Bamacan: a five domain structure including coiled - coil motifs.J Cell Biol, 1997,136:433 - 444.
  • 6Jaworski DM, Kelly GM, Hockfield S. BEHAB, a new member of the proteoglycan tandem repeat family of hyaluronan - binding proteins that is restricted to the brain. J Cell Biol, 1994, 125:495-509.
  • 7Jaworski DM, Kelly GM, Hockfield S. Intracranial injury acutely induces the expression of the secreted isoform of the CNS -specific hyaluronan - binding protein BEHAB/brevican. Exp Neurol, 1999, 157:327 - 337.
  • 8Fraser JRE, Laurent T, Cand UBG, et al.Hyaluronan : its nature, distribution, functions and turnover. J Intern Med, 1997,242:27 - 33.
  • 9Brakebusch C, Seidenbecher CI, Asztely F,et al. Brevican- deficient mice display impaired hippocampal CA1 long - term potentiation but show no obvious deficits in learning and memory. Mol Cell Biol, 2002, 22:7417- 7427.
  • 10Pekny M, Leveen P, Pekna M, et al. Mice lacking glial fibriUary acidic protein displayastrocytes devoid of intermediate filaments but develop and reproduce normally. EMBO J, 1995, 14:1590-1598.

二级参考文献11

  • 1[1]Rauch U, Meyer H, Brakebusch C, et al. Sequence and chromosomal location of the mouse brevican gene. Genomics, 1997, 44:15~21.
  • 2[2]Gray SC, Zerillo CA, Chiang VL, et al. cDNA cloning, chromosomal localization, and expression analysis of human BEHAB/brevican, a brain specific proteoglycan regulated during cortical development and in glioma. Gene, 2000, 256:139~147.
  • 3[3]Sander V, Mullegger J, Lepperdinger G. Xenopus brevican is expressed in the notochord and the brain during early embryogenesis. Mech Dev, 2001 2:251~253.
  • 4[4]Jaworski DM. Kelly GM, Hockfield S. The CNS-specific hyaluronan-binding protein BEHAB is expressed in ventricular zones coincident with gliogenesis. J Neurosci, 1995, 15:1352~1362.
  • 5[5]Ogawa T, Hagihara K, Suzuki M, et al. Brevican in the developing hippocampal fimbria: differential expression in myelinating oligodendrocytes and adult astrocytes suggests a dual role for brevican in central nervous system fiber tract development. J Comp Neurol, 2001 432:285~295.
  • 6[6]Jaworski DM, Kelly GM, Hockfield S. Intracranial injury acutely induces the expression of the secreted isoform of the CNS-specific hyaluronan-binding protein BEHAB/brevican. Exp Neurol, 1999, 157:327~337.
  • 7[7]Jaworski DM, Kelly GM, Piepmeier JM, et al. BEHAB (brain enriched Hyaluronan binding) is expressed in surgical samples of glioma and in intracranial grafts of invasive glioma cell lines. Cancer Res, 1996, 56:2293~2298.
  • 8[8]Matthews RT, Gary SC, Zerillo C, et al. Brain-enriched hyaluronan binding (BEHAB)/brevican cleavage in a glioma cell line is mediated by a disintegrin and metalloproteinase with thrombospondin motifs (ADAMTS) family member. J Biol Chem, 2000, 275:22695~22703.
  • 9[9]Nakamura H, Fujii Y, Inoki I, et al. Brevican is degraded by matrix metalloproteinases and aggrecanase-1 (ADAMTS4) at different sites. J Biol Chem, 2000, 275:38885~38890.
  • 10[10]Nutt CL, Zerillo CA, Kelly GM, et al. Brain enriched hyaluronan binding (BEHAB)/ brevican increases aggressiveness of CNS-1 gliomas in Lewis rats. Cancer Res, 2001, 61:7056~7059.

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