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急性心肌梗死治疗的新看法 被引量:2

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出处 《中国循环杂志》 CSCD 北大核心 2003年第5期323-324,共2页 Chinese Circulation Journal
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  • 1贾志梅,贾志军,周瀛,张利群,王洪淘,齐国先.心肌梗死大鼠非梗死区间质纤维化及机制探讨[J].中国循环杂志,2007,22(5):380-384. 被引量:1
  • 2Zimmermann WH, Cesnjevar R. Cardiac tissue engineering: implications for pediatric heart surgery. Pediatr Cardiol, 2009, 30: 716-723.
  • 3Zhang H, Hou JF, Shen Y, et al. Low level laser irradiation precondition to create friendly milieu of infarcted myocardium and enhance early survival of transplanted bone marrow cells. J Cell Mol Med, 2010, 14: 1975-1987.
  • 4Zhou Q, Zhou JY, Zheng Z, et al. A novel vascularized patch enhances cell survival and modifies ventricular remodeling in a rat myocardial infarction model. J Thorac Cardiovasc Surg, 2010, 140: 1388-1396. el-3.
  • 5Zakharova L, Nural-Guvener H, Nimlos J, et al. Chronic heart failure is associated with transforming growth factor beta-dependent yield and functional decline in atrial explant-derived c-Kit+ cells. J Am Heart Assoc, 2013, 2: e000317.
  • 6Villa MT, Chang DW. Muscle and omental flaps for chest wall reconstruction. Thorac Surg Clin, 2010, 20: 543-550.
  • 7Saifzadeh S, Pourreza B, Hohbenaghi R, et aI. Autogenous greater omentum, as a free nonvascularized graft, enhances bone healing: an experimental nonunion model. J Invest Surg, 2009, 22: 129-137.
  • 8Hughes BG, Schulz R. Targeting MMP-2 to treat ischemic heart injury. Basic Res Cardiol, 2014, 109: 424.
  • 9Spinale FG, Coker ML, Heung LJ, et al. A matrix metalloproteinase induction/activation system exists in the human left ventricular myocardium and is upregulated in heart failure. Circulation, 2000, 102: 1944-1949.
  • 10Li YY, MeTiernan CF, Feldman AM. Interplay of matrix metaHoproteinases, tissue inhihitors of metalloproteinases and their regulators in cardiac matrix remodeling. Cardiovase Res, 2000, 46: 214-224.

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