1[1]Schwartz PJ, Priori SG, Napolitano C. The long QT syndrome[M]. In Zipes DP and Jalife J, (eds.): Cardiac electrophysiology: from cell to bedside. Edited by Third edition W. B. Saunders Company, 2000: pp. 597- 615
8[8]Huikuri H. Dispersion of repolarization and the autonomic system- can we predict torsade de pointes[J ] ?Cardiovasc Drugs Therapy, 2002,16: 93 - 99
9[9]Gold MR, Spencer W. T wave altemans for ventricular arrhythmia risk stratification [ J ]. Curr Opin Cardiol,2003, 18(1) :1 - 5
10[10]Schwartz PJ, Priori SG, Spazzolini C, et al. Genotypephenotype correlation in the long QT syndrome. Gene-specific triggers for life- threatening arrhythmias [J ]. Circulation, 2001, 103:89 - 95
2BezzinaCR RookMB WildeAAM.Cardiac sodium channel and inherited arrhythmia syndromes[J].Cardiovascular Research,2001,49:257-257.
3KyndtF ProbstV PotetF etal.Novel SCNSA mutation leading either to isolated cardiac conduction defect or Brugada syndrome in a large French family [J].Circulation,2001,104:3-3.
5Balser JR. Sodium "channelopathies" and suuden death must you beso sensitive [ J ] ? Circulation Research, 1999,85 : 872.
6Priori SG, Napolitano C, Giordano U,et al. Brugada syndrome and sudden cardiac death in children [ J ]. The Lancent,2000,355 : 808.
7Wang Q, Li ZZ, Shen JX,et al. Genomic organization of the human SCN5A gene encoding the cardiac sodium channel [ J]. Genomics,1996,34:9.
8Grant AO. Molecular biology of sodium channels and their role in cardiac arrhythmias [J]. Am J Med,2001,110:296.
9Wang DW, Viswanathan PC, Balser JR,et al . Clinical, genetic and biophysical characterization of SCN5A mutations associated with atrioventricular conduction block[ J]. Circulation,2002,105:341.
10Akai J, Makita N,Sakurada H,et al . A novel SCNSA mutation associated with idiopathic ventricular fibrrilation without typical ECG findings of Brugada syndrome[ J]. FEBS,2000,479:29.
