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低钠血症诊治新认识 被引量:6

Recent knowledge on diagnosis and therapy of hyponatremia
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出处 《上海医学》 CAS CSCD 北大核心 2003年第11期780-784,共5页 Shanghai Medical Journal
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参考文献33

  • 1Preston GM, Carroll TP, Guggino WB, et al. Appearance of water channels in Xenopus oocytes expressing red cell CHIP28 protein. Science, 1992,256: 385-387.
  • 2Martin PY, Abraham WT, Lieming X, et al. Selective V2-receptor vasopressin-2 antagonism decreases urinary aquaporin-2 excretion in patients with chronic heart failure. J Am Soc Nephrol,1999,10:2165-2170.
  • 3Fujita N, Ishikawa SE, Sasaki S, et al. Role of water channel AQP-CD in water retention in SIADH and cirrhotic rats. Am J Physiol, 1995,269: F926 -F931.
  • 4Fernandez-Llama P, Jimenez W, Bosch-Marce M, et al. Dysregulation of renal aquaporins and Na-Cl cotransporter in CC14-in duced cirrhosis. Kidney Int, 2000,58:216-228.
  • 5Fernandez-Llama P, Turner R, Dibona G, et al. Renal expression of aquaporins in liver cirrhosis induced by chronic common bile duct ligation in rats. J Am Soc Nephrol, 1999, 10: 1950-1957.
  • 6Inoue T, Ohnishi A, Matsuo A, et al. Therapeutic and diagnostic potential of a vasopressin-2 antagonist for impaired water handling in cirrhosis. Clin Pharmacol Ther, 1998,63:561-570.
  • 7Pyo HJ, Summer SN, Niederberger M, et al. Arginine vasopressin gene expression in rats with puromycin-induced nephrotic syndrome. Am J Kidney Dis, 1995,25: 58-62.
  • 8Apostol E, Ecelbarger CA, Terris J, et al. Reduced renal medullary water channel expression in puromycin aminonucleoside-induced nephrotic syndrome. J Am Soc Nephrol, 1997,8:15-24.
  • 9Fernandez-Llama P, Andrews P, Nielsen S, et al. Impaired aquaporin and urea transporter expression in rats with adriamycininduced nephrotic syndrome. Kidney Int,1998,53:1244-1253.
  • 10Saito T, Ishikawa S, Abe K, et al. Acute aquaresis by the nonpeptide arginine vasopressin (AVP) antagonist OPC-31260 improves hyponatremia in patients with syndrome of inappropriate secretion of antidiuretic hormone (SIADH). J Clin Endocrinol Metab, 1997,82:1054-1057.

二级参考文献15

  • 1Anderson RJ, Chung HM, Kluge R et al. Hyponatremia: a prospective analysis of its epidemiology and the pathogenetic role of vasopressin. Ann Int Med, 1985,102:164
  • 2Ayus JC, Krothapalli RK, Arieff AI. Changinconcepts in treatment of severe symptomatic hyponatremia. Rapid correction and possible relation to central pontine myelinolysis.Am J Med, 1985,78:897
  • 3Gross P,Reimann D, Henschkowski J et al. Treatment of severe hyponatremia:conventional and novel aspects.J Am Soc Nephron, 2001,12:s10
  • 4Sterns RH, Silver SM. Hemodialysis in hyponatremia:Is there a risk? Semin Dial,1990,3:3
  • 5Oh MS, Kim HJ, Carroll HJ. Recommendation for treatment of symptomatic hyponatremia. Nephron,1995,70:143
  • 6Peces R, Ablanedo P, Alvarez J. Central pontine and extrapontine myelinolysis following correction of severe hyponatremia. Nephron,1988,49:160
  • 7Ross EA, Nissenson AR. Acid-base and electrolyte disturbances, in Daugirdas JT(ed):Handbook of Dialysis. Boston: Little Brown, 1994. pp401
  • 8Bender FH. Successful treatment of sever hyponatremia in a patient with renal failure using continuous venovenous hemodialysis. Am J Kidney Dis, 1998, 32:829
  • 9JI Daxi, GONG Dehua, XIE Honglang et al. A retrospective study of continuous renal replacement therapy versus intermittent hemodialysis in severe acute renal failure. Chinese Medical Journal,2001,114:1157.
  • 10Ayus jC, Olivero JJ, Frommer JP. Rapid correction of severe hyponatremia with intravenous hypertonic saline solution. Am J Med,1982,72:43

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