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炎症、基质金属蛋白酶降解、氧化应激在腹主动脉瘤发病机制中的研究进展 被引量:4

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摘要 腹主动脉瘤的发病机制尚无定论,通过对人病变组织及小鼠模型的研究发现,主要的发病机制有基质金属蛋白酶降解机制、动脉壁炎症机制及氧化应激机制。本文回顾了近20年研究其发病机制的国内外文献,主要对这3种机制涉及的分子机制的相关进展进行介绍。
出处 《血管与腔内血管外科杂志》 2015年第1期62-65,共4页 Journal of Vascular and Endovascular Surgery
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