1[1]Weisfelner ME,Gottlieb AB.The role of apoptosis in human epidermal keratinocytes. J Drugs Dermatol, 2003; 2(4): 385～91
2[2]Schuler M, Bossy-Wetzel E, Goldstein JC et al. p53 induces apoptosis by caspase activation through mitochondrial cytochrome C release. J Biol Chem, 2000;275(10): 7337～42
3[3]Sitailo LA, Tibudan SS, Denning MF. Activation of Caspase-9 is required for UV-induced apoptosis of human keratinocytes. J Biol Chem, 2002;277(22): 19346～52
4[4]Anna Ettorre, Marco Andreassi, Cecilia Anselmi et al. Involvement of oxidative stress in apoptosis induced by a mixture of isothiazolinones in normal human keratinocytes. Journal of Investigative Dermatology, 2003;121(3):328～36
5[5]Wehrli P, Viard I, Bullani R et al. Death receptors in cutaneous biology and disease. J Invest Dermatol, 2000;115(2): 141～8
6[6]Takahashi H, Ishida-Yamamoto A, Iizuka H. Ultraviolet B irradiation induces apoptosis of keratinocytes by direct activation of Fas antigen. J Investig Dermatol Symp Proc, 2001;6(1):64～8
7[7]Basile JR, Eichten A, Zacny V et al. NF-κB-mediated induction of p21Cip1/Waf1 by tumor necrosis factor a induces growth arrest and cytoprotection in normal human keratinocytes. Mol Cancer Res, 2003;1(4):262～70
8[8]Wang HQ, Quan T, He TY et al. Epidermal growth factor receptor-dependent, NF-κB independent activation of PI-3-kinase/Akt pathway inhibits ultraviolet irradiation-induced Caspases 3, 8, and 9 in human keratinocytes. J Biol Chem, 2003;278(46):45737～45
9[9]Liu X, Alexander V, Vijayachandra K et al. Conditional epidermal expression of TGF-beta 1 blocks neonatal lethality but causes a reversible hyperplasia and alopecia. Proc Nat Acad Sci USA, 2001; 98(16): 9139～44
10[10]Cataisson C, Joseloff E, Murillas R et al. Activation of cutaneous protein kinase C induces keratinocyte apoptosis and intraepidermal inflammation by independent signaling pathways. J Immunol, 2003;171(5): 2703～13
