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心肌梗死后心脏胶原重塑的意义及干预研究 被引量:5

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摘要 心肌梗死后心脏发生修复性纤维化和反应性纤维化等胶原重塑改变 ,参与心力衰竭的发生、发展。针对病理性胶原重塑的深入研究为防治心肌梗死后心力衰竭的发生、发展开辟了新的思路。
作者 吴强 马星卫
出处 《中国循环杂志》 CSCD 北大核心 2003年第6期474-475,共2页 Chinese Circulation Journal
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参考文献15

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同被引文献22

  • 1MinLi Geng-TaoLiu.Inhibition of Fas/FasL mRNA expression and TNF-a release in concanavalin A-induced liver injury in mice by bicyclol[J].World Journal of Gastroenterology,2004,10(12):1775-1779. 被引量:23
  • 2李丹,范哲,李广生.心肌纤维化的调控因素[J].心血管病学进展,2004,25(4):253-257. 被引量:24
  • 3李秀昌,李素梅,张运,张梅,王荣,胡燕燕.三维超声心动图评价心肌梗死患者左室局部心功能[J].中国超声医学杂志,2005,21(4):270-273. 被引量:15
  • 4杨文奇,毛淑丹,陶贵周.急性心肌梗塞后左心室重构的研究现状[J].中国误诊学杂志,2005,5(10):1821-1823. 被引量:9
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  • 7OGATA T, MIYAUCHI T, SAKAI S, et al. Myocardial fibrosis and diastolic dysfunction in deoxycorticosterone acetate-salt hypertensive rats is ameliorated by the peroxisome proliferator-activated receptor-alpha activator fenofibrate, partly by suppressing inflammatory responses associated with the nuclear factor-kappa-B pathway[J]. J Am Coll Cardiol, 2004,43: 1484-1488.
  • 8OGATA T, MIYAUCHI T, SAKAI S, et al. Stimulation of peroxisome-proliferator-activated receptor alpha (PPAR alpha) attenuates cardiac fibrosis and endothelin-1 production in pressure-overloaded rat hearts[J]. Clin Sci (Lond), 2002, 103: 284-288.
  • 9CHEN K, CHEN J, LI D, et al. Angiotensin Ⅱ regulation of collagen type Ⅰ expression in cardiac fibro-blasts: modulation by PPAR-gamma ligand pioglitazone[J]. Hypertension, 2004, 44: 655-661.
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