2Major AS, Dove DE, Ishiguro H, et al. Increased cholesterol efflux in apolipoprotein AI (ApoAI)-producing macrophages as a mechanism for reduced atherosclerosis in ApoAI((-/-)) mice [J]. Arterioscler Thromb Vasc Biol, 2001, 21(11) : 1790-1795.
3Shah PK, Yano J, Reyes O, et al. High-dose recombinant apolipoprotein A-I(milano) mobilizes tissue cholesterol and rapidly reduces plaque lipid and macrophage content in apolipoprotein e-deficient mice. Potential implications for acute plaque stabilization[J]. Circulation, 2001, 103(25): 3047-3050.
4Lee M, Kim JQ, Kim J, et al. Studies on the plasma lipid profiles, and LCAT and CETP activities according to hyperlipoproteinemia phenotypes (HLP) [J]. Atherosclerosis, 2001, 159(2) : 381-389.
5Wang N, Silver DL, Costet P, et al. Specific binding of ApoA-I, enhanced cholesterol efflux, and altered plasma membrane morphology in cells expressing ABC1 [J]. J Biol Chem, 2000, 275 (42) : 33053-33058.
6Oram JF, Lawn RM, Garvin MR, et al. ABCA1 is the cAMP-inducible apolipoprotein receptor that mediates cholesterol secretion from macrophages[J]. J Biol Chem, 2000, 275 (44): 34508-34511.
7Navab M, Hama SY, Cooke CJ, et al, Normal high density lipoprotein inhibits three steps in the formation of mildly oxidized low density lipoprotein: step 1[J], J Lipid Res, 2000, 41(9): 1481-1494.
8Navab M, Hama SY, Anantharamaiah GM, et al. Normal high density lipoprotein inhibits three steps in the formation of mildly oxidized low density lipoprotein: steps 2 and 3[J]. J Lipid Res, 2000, 41(9): 1495-1508.
9Rodrigo L, Mackness B, Durrington PN, et al. Hydrolysis of platelet-activating factor by human serum paraoxonase[J]. Bio-chem J, 2001, 354(Pt 1): 1-7.
10Cockerill GW, Huehns TY, Weerasinghe A, et al. Elevation of plasma high-density lipoprotein concentration reduces interleukin-1-induced expression of E-selectin in an in vivo model of acute inflammation [J]. Circulation, 2001, 103(1): 108-112.