1[1]Patel DJ,Knight CJ,Holdright DR,et al.Pathophysiology of transient myocardial ischemia in acute coronary syndromes:characterization by continuous ST-segment monitoring[J].Circulation,1997,95:1185.
2[2]Cannon CP,Mocabe CH,Stone PH,et al.for the TIMI Ⅲregistry and TIMI ⅢB investigators circadian:variation in the onset of unstable angina and non-Q wave acute myocardial infarction[J].Am J Cardiol,1997,79:253.
3[3]Tummala PE,Chen XL,Sundell CL,et al.Angiotensin Ⅱ induces vascular cell adhesion molecule-1 expression in rat vasculature: a potential link between the rennin-angiotensin system and atherosclerosis[J].Circ Res,1999,100:1223.
4[4]Piqueas L,Kubes P,Alvarez A,et al.Angiotensin Ⅱ induces leukocyte-endothelial cell interactions in vivo via AT1 and AT2 receptor-mediated P-selectin uprugulation[J].Circulation,2000,102:2118.
5[5]Xi-Lin Chen,Pradyumna E,Tummala,et al.Angiotensin Ⅱ induces monocyte chemoattractant protein-1 gene expression in rat vascular smooth muscle cells[J].Circ Res,1998,83: 952.
6[6]Nakamura A,Johns EJ,Imaizumi A,et al.Effect of beta(2)-adrenoceptor activation and angiotensin Ⅱ on tumour necrosis factor and interleukin 6 gene transcription in the rat renal resident macrophage cells[J].Cytokine,1999,11(10):759.
7[7]Phillips MI,Kagiyama S.AngiotensinⅡas a pro-inflammatory mediator[J].Curr Opin Investig Drugs,2002,3(4):569.
8[8]Moreno PR,Falk E,Palacios IF,et al.Macrophage infiltration in acute coronary syndromes.Implications for plaque rupture[J].Circulation,1994,90:775.
9[9]Libby P.Molecular bases of the acute coronary syndromes[J].Circulation,1995,91:2844.
10[10]Kovanen PT,Kaartinen M,Paavonen T.Infiltrates of activated mast cells at the site of coronary atheromatous erosion or rupture in myocardial infarction[J].Circulation,1995,92:1084.