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丙型肝炎病毒对磷脂酶A2基因表达的调节

Regulation of the expression of phospholipase A2 gene
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作者 纪冬 成军
机构地区 解放军第
出处 《胃肠病学和肝病学杂志》 CAS 2004年第1期94-96,共3页 Chinese Journal of Gastroenterology and Hepatology
基金 ~~
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参考文献22

  • 1Milella M, Gismondi A, Roncaioli P, et al. CD16 cross-linking induces both secretory and extracellular signal-regulated kinase (ERK)-dependent cytosolic phospholipase A2(PLA2) activity in human natural killer cells: involvement of ERK,but not PLA2, in CD16-triggered granule exocytosis. J Immunol, 1997,158(7) :3148-3154.
  • 2Dennis EA. Diversity of group types, regulation and function of phospholipase A2.J Biol Chem, 1994,269(18): 13057-130606.
  • 3Balboa MA, Shirai Y, Gaietta G, et al. Localization of group V phospholipase A2 in caveolin-enriched granules in activated P388D1 macrophagelike cells. J Biol Chem, 2003,278 (48) :48059-48065.
  • 4Tischfield JA. A reassessment of the low molecular weight phospholipase A2 gene family in mammals. J Biol Chem, 1997,272(28):17247-17250.
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  • 6Nevalainen TJ. Serum phospholipase A2 in inflammatory diseases. Clin Chem, 1993,39(12): 2453-2459.
  • 7Lin MK, Farewell V, Vadas P, et al. Secretory phospholipase A2 as an index of disease activity in rheumatoid arthritis. Prospective double blind study of 212 patients .J Rheumatol, 1996,23(7): 1162-1166.
  • 8Tietge UJ, Maugeais C,Lund-Katz S, et al. Human secretory phospholipase A2 mediates decreased plasma levels of HDL cholesterol and apoA- Ⅰ in response to inflammation in human apoA- Ⅰ transgenic mice. Arterioscler Thromb Vasc Biol,2002,22(7): 1213-1218.
  • 9Touqui L,Alaoui-E1-Azher M.Mammalian secreted phospholipases A2 and their pathophysiological significance in inflammatory diseases. Curr Mol Med,2001,1(6) :739-754.
  • 10Beck S, Lambeau G, Scholz-Pedretti K, et al. Potentiation of tumor necrosis factor alpha-induced secreted phospholipase A2 ( sPLA2 )- Ⅱ A expression in mesangial cells by an autocrine loop involving sPLA2 and peroxisome proliferator-activated receptor alpha activation. J Biol Chem, 2003, 278(32): 29799-29812.

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