Protein chemistry determines light chain-mediated renal damage in patients with plasma cell dyscrasias: a molecular understanding of variable manifestations
1MacIntire W.Case of mollities and fragilis ossium accompanied by urine strongly charged with animal matter,1850.
2Bence Jones H.On a new substance occurring in the urine of a patient with "mollitis ossium"[J],1848.
3Wright JH.A case of multiple myeloma,1900.
4Krauss E.Studien zur Bence-Jones′schen Albuminurie,1899.
5Magnus-Levy A.Bence-Jones-Elweiss und Amyloid,1931.
6Bell ET.Renal lesions associated with multiple myeloma,1933.
7Glenner GG;Terry W;Harada M.Amyloid fibril proteins:proof of homology with immunoglobulin light chains by sequence analyses[J],1971.
8Randall RE;Williamson WC Jr;Mullinax F.Manifestations of systemic light chain deposition[J],1976.
9Sanders PW;Herrera GA;Chen A.Differential nephrotoxicity of low molecular weight proteins including Bence Jones proteins in the perfused rat nephron in vivo[J],1988.
10Sanders PW;Herrera GA;Lott RL.Morphologic alterations of the proximal tubules in light chain-related renal disease[J],1988.
5Kindy MS, De Beer FC. Amyloidosis. In:Massry SG, Glassock RJ, eds. Textbook of nephrology. 4th ed. Philadephia: Lippincott Williams and wilkins,2001.824-829.
6Amyloidosis. In: Kern WF, Silva FG, Laszik ZG, et al, eds. Atlas of renal pathology. Philadephia: W.B.Saunders Company,1999.221-228.
7Falk RH, Comenzo RL, Skinner M. He systemic amyloidoses.N Engl J Med,1997, 337:898-909.
