关于无氧阈机制的人体与动物实验对比研究

Experimental Contrastive Study between the Human Body and Animal Level on the Mechanism of "Anaerobic Threshold" Theory

运动中体内的糖代谢由有氧氧化向糖酵解转变的机制问题,过去一直认为是缺氧造成的。近十年来,在运动生理学界有大量实验证明转变时体内并不缺氧,故引起对"无氧阈"概念准确性和转变机制的深入探讨。提出运动中有氧产能过程与当时耗能过程不匹配是代谢转变机制的原因;而丙酮酸转变成乳酸是为了防止其堆积对糖酵解产能过程的抑制作用,以保证其快速供能,与缺氧与否无关的理论假说。依据文献资料,抓住转变时生理生化的机理分析,从整体角度出发进行了人体与动物实验的对比研究,以证明该假说的信度和效度,为今后深入研究打下基础。

In the past,it had been considered that anoxia caused the conversion of glycogen of body during exercise from aerobic oxidation to glycolysis.In recent ten years,many experiments in the sports physiology field have proved that the body during exercise was not lack of oxygen when aerobic metabolism converts to glycolysis,so researchers begin to make a thorough discussion on the accuracy of 'anaerobic threshold' and the conversion's mechanism.This study suggests that the reason of conversion is that the process of energy production under the aerobic condition does not match with the process of energy consumption during the exercise.The purposes of conversion from pyruvate to lactate are to prevent the inhibiting effect of pyruvate accumulation on the process of energy production of glycolysis and to provide energy fast.The conversion's mechanism has nothing to do with the anoxia.This experiment was based on the literatures and analysis according to the physiochemical mechanism of the conversion,and the human body and animal experimental study was carried out from the whole point of view.The purposes were to prove the creditability and validity of the assumption and to give a base to the further study.