孤立性脑桥梗死与脑白质高信号的研究进展

Advances in Research on Isolated Pontine Infarction and White Matter Hyperintensity

脑桥的解剖结构较为复杂,基底动脉(Basilar artery, BA)和脑桥穿支动脉是主要的供血动脉。孤立性脑桥梗死(pontine infarction, PI)的发病机制可由脑桥穿支病变所致,目前机制仍在研究中。脑白质高信号(white matter hyperintensity, WMH)是脑小血管病的重要影像标记物之一,而且穿支血管与小血管有更紧密的联系,WMH可能与穿支供血区梗死患者的穿支动脉病变关系密切。目前PI与WMH之间的关系仍在研究中,现就PI与WMH的关系作相关的综述。

The anatomical structure of the pons is complex, and the basilar artery (BA) and pontine perforating arteries are the main blood supply arteries. The pathogenesis of isolated pontine infarction (PI) can be caused by pontine perforator lesions, and the mechanism is still under study. White matter hyperintensity (WMH) is one of the important imaging markers of cerebral small vessel disease, and perforator vessels are more closely related to small vessels, WMH may be closely related to the lesion of perforator artery in patients with infarction in perforator area. At present, the relationship between PI and WMH is still under study. Now, we will review the relationship between PI and WMH.