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Numerical investigation on threading dislocation bending with InAs/GaAs quantum dots
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作者 Guo-Feng Wu Jun Wang +8 位作者 Wei-Rong Chen Li-Na Zhu Yuan-Qing Yang Jia-Chen Li chun-yang xiao Yong-Qing Huang xiao-Min Ren Hai-Ming Ji Shuai Luo 《Chinese Physics B》 SCIE EI CAS CSCD 2021年第11期146-150,共5页
The threading dislocations(TDs)in GaAs/Si epitaxial layers due to the lattice mismatch seriously degrade the performance of the lasers grown on silicon.The insertion of InAs quantum dots(QDs)acting as dislocation filt... The threading dislocations(TDs)in GaAs/Si epitaxial layers due to the lattice mismatch seriously degrade the performance of the lasers grown on silicon.The insertion of InAs quantum dots(QDs)acting as dislocation filters is a pretty good alternative to solving this problem.In this paper,a finite element method(FEM)is proposed to calculate the critical condition for InAs/GaAs QDs bending TDs into interfacial misfit dislocations(MDs).Making a comparison of elastic strain energy between the two isolated systems,a reasonable result is obtained.The effect of the cap layer thickness and the base width of QDs on TD bending are studied,and the results show that the bending area ratio of single QD(the bending area divided by the area of the QD base)is evidently affected by the two factors.Moreover,we present a method to evaluate the bending capability of single-layer QDs and multi-layer QDs.For the QD with 24-nm base width and 5-nm cap layer thickness,taking the QD density of 10^(11) cm^(-2) into account,the bending area ratio of single-layer QDs(the area of bending TD divided by the area of QD layer)is about 38.71%.With inserting five-layer InAs QDs,the TD density decreases by 91.35%.The results offer the guidelines for designing the QD dislocation filters and provide an important step towards realizing the photonic integration circuits on silicon. 展开更多
关键词 InAs/GaAs quantum dots threading dislocation finite element method bending area
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C/EBPβ控制耐力运动诱发的心脏生理性生长和抗病理性重构
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作者 Pontus Bostrm Nina Mann +8 位作者 Jun Wu Pablo A Quintero Eva R Plovie Daniela Panáková Rana K Gupta chun-yang xiao Calum A MacRae Anthony Rosenzweig Bruce M Spiegelman 《中南医学科学杂志》 CAS 2013年第5期519-519,共1页
心肌能通过使其细胞增大来适应外界压力的增加,这一应激反应适应于生理性刺激或病理性刺激对心脏造成的高负荷压力。例如,在负荷运动,高血压等刺激下,心肌都会出现明显的肥大和增殖现象。病理性心肌肥大可导致心力衰竭或心律失常,... 心肌能通过使其细胞增大来适应外界压力的增加,这一应激反应适应于生理性刺激或病理性刺激对心脏造成的高负荷压力。例如,在负荷运动,高血压等刺激下,心肌都会出现明显的肥大和增殖现象。病理性心肌肥大可导致心力衰竭或心律失常,但生理性肥厚却不能引起这些疾病,所以弄清楚这两种类型的心肌肥大分子机制具有重要的临床意义。 展开更多
关键词 病理性重构 生理性 C EBPβ 运动诱发 心脏 心肌肥大 生长 耐力
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