This study was conducted during 2019/2020 on sweet cherry trees (<i>Prunus Avium</i> L.) (Bing and Hardy Giant) cultivar planted at Sergaya-Al_Zabadani area of Rural </span><span style="font-...This study was conducted during 2019/2020 on sweet cherry trees (<i>Prunus Avium</i> L.) (Bing and Hardy Giant) cultivar planted at Sergaya-Al_Zabadani area of Rural </span><span style="font-family:"">Dam</span><span style="font-family:"">ascus, to reduce fruit drop of sweet cherry. The experiment included 4 foliar applications: T1: control, T2: GA<sub>3</sub> (100 ppm), T3: NAA (20 <span>ppm), T4: (100 ppm GA<sub>3</sub> + 20 ppm NAA). Fruit set and fruit drop pe</span>rcentage, fruiting factor, and yield were recorded. The results showed that treatment with (100 ppm GA<sub>3</sub> + 20 ppm NAA) recorded higher fruit set percentage (73.81% and 75.62%), and fruiting factor (48.38% and 50.04%) respectively</span><span style="font-family:"">;</span><span style="font-family:""> <span>In addition to fruit yield (40.19 and 41.21 kg/tree) for both cultivars, co</span>mpared to the control (9.13, 6.60 kg/tree). Therefore, it can be concluded that GA<sub>3</sub> + NAA treatment reduced Sweet cherry fruit drop better than other treatments, <span>where fruit drop percentage didn’t exceed (63.11% and 62.01%) in both cu</span>ltivars (Bing and Hardy Giant) respectively, compared to the control (80.92% and 80.64%).展开更多
Objective To investigate the effect of naphthalene acetic acid(NAA) on proliferation and apoptosis of mice hepatocytes.Methods Fifty mice were randomly divided into five groups: positive control,normal control,low,mid...Objective To investigate the effect of naphthalene acetic acid(NAA) on proliferation and apoptosis of mice hepatocytes.Methods Fifty mice were randomly divided into five groups: positive control,normal control,low,middle and high dose of NAA groups.Mice in low,middle and high dose of naphthalene acetic acid groups were fed with 200 mg/kg,1 000 mg/kg and 5 000 mg/kg NAA animal food,respectively.Mice in positive control,normal control groups were fed with usual animal food.After 4 weeks,mice were sacrificed and liver was separated.Three days before scarification,mice in positive control group were given 20 mg/kg ascyclophosphami daily by intraperitoneal injection.The proliferative activity of hepatocytes was detected by MTT assay.The expressions of PCNA protein and Caspase-3 were measured by immunohistochemistry method.Results The proliferative activities of mice hepatocytes in high dose group and normal control group were 0.794±0.019 and 1.055±0.019, respectively.The expressions of PCNA protein and Caspase-3 in high dose group were 11.2% and 37.9%,and those in the normal group were 33.1% and 6.3%, respectively.All differences were significant(P<0.01).Conclusion High dose of NAA acid could significantly inhibit proliferation and increase apoptosis of hepatocytes in mice.展开更多
文摘This study was conducted during 2019/2020 on sweet cherry trees (<i>Prunus Avium</i> L.) (Bing and Hardy Giant) cultivar planted at Sergaya-Al_Zabadani area of Rural </span><span style="font-family:"">Dam</span><span style="font-family:"">ascus, to reduce fruit drop of sweet cherry. The experiment included 4 foliar applications: T1: control, T2: GA<sub>3</sub> (100 ppm), T3: NAA (20 <span>ppm), T4: (100 ppm GA<sub>3</sub> + 20 ppm NAA). Fruit set and fruit drop pe</span>rcentage, fruiting factor, and yield were recorded. The results showed that treatment with (100 ppm GA<sub>3</sub> + 20 ppm NAA) recorded higher fruit set percentage (73.81% and 75.62%), and fruiting factor (48.38% and 50.04%) respectively</span><span style="font-family:"">;</span><span style="font-family:""> <span>In addition to fruit yield (40.19 and 41.21 kg/tree) for both cultivars, co</span>mpared to the control (9.13, 6.60 kg/tree). Therefore, it can be concluded that GA<sub>3</sub> + NAA treatment reduced Sweet cherry fruit drop better than other treatments, <span>where fruit drop percentage didn’t exceed (63.11% and 62.01%) in both cu</span>ltivars (Bing and Hardy Giant) respectively, compared to the control (80.92% and 80.64%).
文摘Objective To investigate the effect of naphthalene acetic acid(NAA) on proliferation and apoptosis of mice hepatocytes.Methods Fifty mice were randomly divided into five groups: positive control,normal control,low,middle and high dose of NAA groups.Mice in low,middle and high dose of naphthalene acetic acid groups were fed with 200 mg/kg,1 000 mg/kg and 5 000 mg/kg NAA animal food,respectively.Mice in positive control,normal control groups were fed with usual animal food.After 4 weeks,mice were sacrificed and liver was separated.Three days before scarification,mice in positive control group were given 20 mg/kg ascyclophosphami daily by intraperitoneal injection.The proliferative activity of hepatocytes was detected by MTT assay.The expressions of PCNA protein and Caspase-3 were measured by immunohistochemistry method.Results The proliferative activities of mice hepatocytes in high dose group and normal control group were 0.794±0.019 and 1.055±0.019, respectively.The expressions of PCNA protein and Caspase-3 in high dose group were 11.2% and 37.9%,and those in the normal group were 33.1% and 6.3%, respectively.All differences were significant(P<0.01).Conclusion High dose of NAA acid could significantly inhibit proliferation and increase apoptosis of hepatocytes in mice.
