Background: A decrease in trabecular cell number has been reported in treated glaucoma patients. The mechanisms of this cell loss are poorly understood. This study evaluates the effect of benzalkonium-preserved (BAC+)...Background: A decrease in trabecular cell number has been reported in treated glaucoma patients. The mechanisms of this cell loss are poorly understood. This study evaluates the effect of benzalkonium-preserved (BAC+) or preservative-free (BAC-) antiglaucoma medications on expression of apoptotic markers expression by cultured human trabecular cells to see whether apoptosis may be one mechanism by which trabecular cells.展开更多
Purpose: Antiglaucoma drugs have been associated with con-junctival and trabecular inflammatory cell infiltrates. However, the underlying mechanisms are still poorly understood. The aim of this study was to assess the...Purpose: Antiglaucoma drugs have been associated with con-junctival and trabecular inflammatory cell infiltrates. However, the underlying mechanisms are still poorly understood. The aim of this study was to assess the effects of antiglaucoma medica-展开更多
文摘Background: A decrease in trabecular cell number has been reported in treated glaucoma patients. The mechanisms of this cell loss are poorly understood. This study evaluates the effect of benzalkonium-preserved (BAC+) or preservative-free (BAC-) antiglaucoma medications on expression of apoptotic markers expression by cultured human trabecular cells to see whether apoptosis may be one mechanism by which trabecular cells.
文摘Purpose: Antiglaucoma drugs have been associated with con-junctival and trabecular inflammatory cell infiltrates. However, the underlying mechanisms are still poorly understood. The aim of this study was to assess the effects of antiglaucoma medica-
