Background: A decrease in trabecular cell number has been reported in treated glaucoma patients. The mechanisms of this cell loss are poorly understood. This study evaluates the effect of benzalkonium-preserved (BAC+)...Background: A decrease in trabecular cell number has been reported in treated glaucoma patients. The mechanisms of this cell loss are poorly understood. This study evaluates the effect of benzalkonium-preserved (BAC+) or preservative-free (BAC-) antiglaucoma medications on expression of apoptotic markers expression by cultured human trabecular cells to see whether apoptosis may be one mechanism by which trabecular cells.展开更多
Purpose: To assess, by flow cytometry, the expressions of inflammatory markers HLA-DR, IL-6 and IL-8, by conjunctival epithelial cells obtained by impression cytology from long-term treated glaucoma patients and norma...Purpose: To assess, by flow cytometry, the expressions of inflammatory markers HLA-DR, IL-6 and IL-8, by conjunctival epithelial cells obtained by impression cytology from long-term treated glaucoma patients and normal subjects. Methods: 64 impression cytology samples were collected from 54 patients: CD 39 patients suffering from POAG and receiving prolonged展开更多
文摘Background: A decrease in trabecular cell number has been reported in treated glaucoma patients. The mechanisms of this cell loss are poorly understood. This study evaluates the effect of benzalkonium-preserved (BAC+) or preservative-free (BAC-) antiglaucoma medications on expression of apoptotic markers expression by cultured human trabecular cells to see whether apoptosis may be one mechanism by which trabecular cells.
文摘Purpose: To assess, by flow cytometry, the expressions of inflammatory markers HLA-DR, IL-6 and IL-8, by conjunctival epithelial cells obtained by impression cytology from long-term treated glaucoma patients and normal subjects. Methods: 64 impression cytology samples were collected from 54 patients: CD 39 patients suffering from POAG and receiving prolonged
