Introduction Autoimmune diseases result from a breach of immunological self-tolerance and tissue damage by autoreactive T lymphocytes.Severe acute respiratory syndrome coronavirus-2(SARS-CoV-2)infection is characteriz...Introduction Autoimmune diseases result from a breach of immunological self-tolerance and tissue damage by autoreactive T lymphocytes.Severe acute respiratory syndrome coronavirus-2(SARS-CoV-2)infection is characterized by an inflammatory dysregulation that has been associated with the development of autoimmune processes[1].展开更多
基金Instituto de Investigacion Biom-edica de Malaga-IBIMA,Hospital Universitario Virgen de la Victoria,Universidad de Malaga,Malaga,Spain,Centro de Investigacion Biomedica en Red de Enfermedades Hepa ticas y Digestivas(CIBERehd),Madrid,SpainGrants of Instituto de Salud CarlosⅢcofounded by Fondo Europeo de Desarrollo Regional-FEDER[contract numbers:PI18/00901,UMA18-FEDERJA-193],COST ACTION CA17112-Prospective European Drug-Induced Liver Injury Network and IMI2-Translational Safety Biomarker Pipeline(TransBioLine Horizonte 2020)CM21/00074(Rio Hortega contract:J.M.P.B.).
文摘Introduction Autoimmune diseases result from a breach of immunological self-tolerance and tissue damage by autoreactive T lymphocytes.Severe acute respiratory syndrome coronavirus-2(SARS-CoV-2)infection is characterized by an inflammatory dysregulation that has been associated with the development of autoimmune processes[1].
