在过去10年里,基于量子力学的量子信号表示研究已经出现一些重要结果.然而,关于量子信号处理方面的研究却相对滞后,其中就包括量子信号的滤波处理.首先,改进了现有的数字信号的量子表示模型(quantum representation of digital signals,...在过去10年里,基于量子力学的量子信号表示研究已经出现一些重要结果.然而,关于量子信号处理方面的研究却相对滞后,其中就包括量子信号的滤波处理.首先,改进了现有的数字信号的量子表示模型(quantum representation of digital signals,QRDS),使其适用于任意长度的时间信号,同时还修改了QRDS模型中二补码的编码方法,使得新的编码更符合实际问题.然后,基于改进的模型引入了中值滤波方案,该方案回避了量子计算不能直接实现卷积运算的缺陷.为了实现该滤波方案的量子电路,又给出了基本量子运算模块:比较器模块、交换模块和中值计算模块.最后,通过实例验证了文中所提滤波方案的有效性和合理性.展开更多
To clarify the possible mechanisms of α anordin and probimane on calmodulin Ca^(++)-Mg^(++)-ATPase system, enzyme dynamic study was carried out by determining three dynamic parameters [the substrate concentration(AT...To clarify the possible mechanisms of α anordin and probimane on calmodulin Ca^(++)-Mg^(++)-ATPase system, enzyme dynamic study was carried out by determining three dynamic parameters [the substrate concentration(ATP) response curve, dose(inhibitors) response curve and time response curve]. Our data have shown that the inhibitory rates of α anordrin and probimane are unrelated to substrate(APT) concentrations, but related to calmodulin concentrations. The inhibition of α anordrin and probimane is very quick that is completed within 1 to 5 min and can maintain more than 1 hr in the same inhibitory rates. So it is possible that α anordrin and probimane are calmodulin competitors with calmodulin like binding whose actions can occur by affecting the reaction balance of substrate and product on target enzymes of calmodulin( Ca^(++)-Mg^(++)-ATPase).展开更多
To broaden the applications of magnetized water(MW) in medical science, the possible detoxicative effect of MW to anticancer drugs in vivo were studied. After being given ip with cyclophosphomide (CTX) 500 mg...To broaden the applications of magnetized water(MW) in medical science, the possible detoxicative effect of MW to anticancer drugs in vivo were studied. After being given ip with cyclophosphomide (CTX) 500 mg/kg, cisplatin (DDP) 40 mg/kg, harringtonine (HA) 20 mg/kg, mitomycin C (MMC) 8 mg/kg, lycobetaine (Lyc) 200 mg/kg, respectively, the mice were given MW ip 0.2 ml for 7 days. The average life span was calculated for each group. After being given subacutely lower doses of anticancer drugs ( CTX 100 mg/kg, HA 3 mg/kg ) ip 3 times, the mice were given MW ip 0.2 ml for 7 days and the blood white cells were counted as routine. It was shown that the mice in MW groups after ip anticancer drugs survived longer than those without MW. The effects of various anticancer drugs on life span were different. The white cell numbers of groups with MW were higher than that of the groups without MW. So it is possible that MW can remarkably extend the life span of mice and attenuate the leukopenia by mitigating the toxicity of anticancer drugs in vivo .展开更多
文摘在过去10年里,基于量子力学的量子信号表示研究已经出现一些重要结果.然而,关于量子信号处理方面的研究却相对滞后,其中就包括量子信号的滤波处理.首先,改进了现有的数字信号的量子表示模型(quantum representation of digital signals,QRDS),使其适用于任意长度的时间信号,同时还修改了QRDS模型中二补码的编码方法,使得新的编码更符合实际问题.然后,基于改进的模型引入了中值滤波方案,该方案回避了量子计算不能直接实现卷积运算的缺陷.为了实现该滤波方案的量子电路,又给出了基本量子运算模块:比较器模块、交换模块和中值计算模块.最后,通过实例验证了文中所提滤波方案的有效性和合理性.
文摘To clarify the possible mechanisms of α anordin and probimane on calmodulin Ca^(++)-Mg^(++)-ATPase system, enzyme dynamic study was carried out by determining three dynamic parameters [the substrate concentration(ATP) response curve, dose(inhibitors) response curve and time response curve]. Our data have shown that the inhibitory rates of α anordrin and probimane are unrelated to substrate(APT) concentrations, but related to calmodulin concentrations. The inhibition of α anordrin and probimane is very quick that is completed within 1 to 5 min and can maintain more than 1 hr in the same inhibitory rates. So it is possible that α anordrin and probimane are calmodulin competitors with calmodulin like binding whose actions can occur by affecting the reaction balance of substrate and product on target enzymes of calmodulin( Ca^(++)-Mg^(++)-ATPase).
文摘To broaden the applications of magnetized water(MW) in medical science, the possible detoxicative effect of MW to anticancer drugs in vivo were studied. After being given ip with cyclophosphomide (CTX) 500 mg/kg, cisplatin (DDP) 40 mg/kg, harringtonine (HA) 20 mg/kg, mitomycin C (MMC) 8 mg/kg, lycobetaine (Lyc) 200 mg/kg, respectively, the mice were given MW ip 0.2 ml for 7 days. The average life span was calculated for each group. After being given subacutely lower doses of anticancer drugs ( CTX 100 mg/kg, HA 3 mg/kg ) ip 3 times, the mice were given MW ip 0.2 ml for 7 days and the blood white cells were counted as routine. It was shown that the mice in MW groups after ip anticancer drugs survived longer than those without MW. The effects of various anticancer drugs on life span were different. The white cell numbers of groups with MW were higher than that of the groups without MW. So it is possible that MW can remarkably extend the life span of mice and attenuate the leukopenia by mitigating the toxicity of anticancer drugs in vivo .
