不透水层是指能够隔离地表水渗透到土壤的覆盖表面,以不透水层分布变化来研究铜川城市化进程。利用决策树分类结合生物物理成分指数(BCI)和裸土指数(MBSI)的方法对1986,1991,1996,2002,2007,2012和2017年的遥感影像数进行不透水层提取,...不透水层是指能够隔离地表水渗透到土壤的覆盖表面,以不透水层分布变化来研究铜川城市化进程。利用决策树分类结合生物物理成分指数(BCI)和裸土指数(MBSI)的方法对1986,1991,1996,2002,2007,2012和2017年的遥感影像数进行不透水层提取,采用指标分析、重心轨迹偏移等方法探究不透水层空间扩展特征,并结合统计年鉴、DEM数据研究铜川市不透水层扩展驱动机制。结果表明:文中提出的基于决策树分类模型结合BCI和MBSI的方法对不透水层提取与验证数据的拟合优度达到0.88.铜川市的不透水层面积持续增加,面积从1986年的5.7 km 2增加到2017年的61.5 km 2,年均增长速度高达1.8%,特别是2007—2017年是快速城市化时期,增长面积占31 a变化总面积的69.3%.不透水层的重心呈先北后南的阶段性变化,1986—2002年向北移动,2002—2007年向南移动,2007—2017年继续向西南方向移动。通过对驱动力指标分析表明,经济及人口增长对不透水层扩展有着直接推动作用,矿产资源分布、地理环境限制和规划政策引导为影响研究区不透水层变化的主要因素。展开更多
Trichloropropane（TCP） is a chlorinated solvent which derives from chemical manufacturing as a precursor, and it is also an important constituent of solvent formulations in cleaning/degreasing operations. The control...Trichloropropane（TCP） is a chlorinated solvent which derives from chemical manufacturing as a precursor, and it is also an important constituent of solvent formulations in cleaning/degreasing operations. The control and remediation of TCP in polluted sites is a challenge for many conventional remediation techniques due to its refractory behaviour. This challenge in mind, some nano-materials and oxidants were tested to evaluate their effectiveness as in TCP degradation in a laboratory setting. Experimental results indicate that the use of nanoscale zero-valent iron prepared by green tea（GT） as a reductant has negligible degradation effect on TCP under normal temperature and pressure conditions. However, zinc powders of similar size but higher surface reactivity, demonstrated stronger dechlorination capacity in the breakdown of TCP, as almost all of TCP was degraded by carboxymethocel（CMC） stabilized nanoscale zinc within 24 h. Activated persulfate by citric acid（CA） and chelated Fe（Ⅱ） was also used for TCP treatment with a TCP removal efficiency rate of nearly 50% within a 24 h reaction period, and a molar ratio of S2O82-, Fe2+ and CA is 20:5:1. Both the reduction and oxidation reactions are in accordance with the pseudo-first order kinetic equation. These results are promising for future use of TCP for the remediation of polluted sites.展开更多
Background Inflammation and immunity play a vital role in the pathogenesis of early brain injury after subarachnoid hemorrhage （SAH）. Nuclear factor-kappa B （NF-κB） regulates many genes essential for inflammation...Background Inflammation and immunity play a vital role in the pathogenesis of early brain injury after subarachnoid hemorrhage （SAH）. Nuclear factor-kappa B （NF-κB） regulates many genes essential for inflammation and immunity and is activated by toll-like receptor （TLR）. This study aimed to detect the expression of the toll-like receptor 4/nuclear factor-kappa B （TLR4/NF-κB） signaling in the rat brain after early SAH. Methods The rats were decapitated and their brains were removed at 0, 2, 4, 6, 12, 24 and 48 hours after a single injection of blood into the prechiasmatic cistern, mRNA expression of TLR4 was measured by Taqman real-time RT-PCR, and protein expression by immunohistochemistry and Western blotting. NF-κB activity and concentrations of tumor necrosis factor-alpha （TNF-α）, interleukin-lbeta （IL-1β） and interleukin-6 （IL-6） were measured by enzyme-linked immunosorbent assay （ELISA）. Results TaqMan real-time RT-PCR and Western blotting identified a biphasic change in TLR4 expression in both mRNA and protein： an initial peak （2-6 hours） and a sustained elevation （12-48 hours）. Immunohistochemical staining showed the inducible expression of TLR4-like immunoreactions predominantly in glial cells and vascular endothelium. A similar biphasic change in the activation of NF-κB subunit p65 as well as the production of NF-κB-regulated proinflammatory cytokines （TNF-α, IL-1β and IL-6） were detected by ELISA. Conclusions These data suggest that experimental SAH induces significant up-regulation of TLR4 expression and the NF-κB signaling in early brain injury. Activation of the TLR4/NF-κB signaling may regulate the inflammatory responses after SAH.展开更多
文摘不透水层是指能够隔离地表水渗透到土壤的覆盖表面,以不透水层分布变化来研究铜川城市化进程。利用决策树分类结合生物物理成分指数(BCI)和裸土指数(MBSI)的方法对1986,1991,1996,2002,2007,2012和2017年的遥感影像数进行不透水层提取,采用指标分析、重心轨迹偏移等方法探究不透水层空间扩展特征,并结合统计年鉴、DEM数据研究铜川市不透水层扩展驱动机制。结果表明:文中提出的基于决策树分类模型结合BCI和MBSI的方法对不透水层提取与验证数据的拟合优度达到0.88.铜川市的不透水层面积持续增加,面积从1986年的5.7 km 2增加到2017年的61.5 km 2,年均增长速度高达1.8%,特别是2007—2017年是快速城市化时期,增长面积占31 a变化总面积的69.3%.不透水层的重心呈先北后南的阶段性变化,1986—2002年向北移动,2002—2007年向南移动,2007—2017年继续向西南方向移动。通过对驱动力指标分析表明,经济及人口增长对不透水层扩展有着直接推动作用,矿产资源分布、地理环境限制和规划政策引导为影响研究区不透水层变化的主要因素。
基金supported by Basal Science Research Fund from the Chinese Academy of Geological Sciences(Grant No.YWF201405)
文摘Trichloropropane（TCP） is a chlorinated solvent which derives from chemical manufacturing as a precursor, and it is also an important constituent of solvent formulations in cleaning/degreasing operations. The control and remediation of TCP in polluted sites is a challenge for many conventional remediation techniques due to its refractory behaviour. This challenge in mind, some nano-materials and oxidants were tested to evaluate their effectiveness as in TCP degradation in a laboratory setting. Experimental results indicate that the use of nanoscale zero-valent iron prepared by green tea（GT） as a reductant has negligible degradation effect on TCP under normal temperature and pressure conditions. However, zinc powders of similar size but higher surface reactivity, demonstrated stronger dechlorination capacity in the breakdown of TCP, as almost all of TCP was degraded by carboxymethocel（CMC） stabilized nanoscale zinc within 24 h. Activated persulfate by citric acid（CA） and chelated Fe（Ⅱ） was also used for TCP treatment with a TCP removal efficiency rate of nearly 50% within a 24 h reaction period, and a molar ratio of S2O82-, Fe2+ and CA is 20:5:1. Both the reduction and oxidation reactions are in accordance with the pseudo-first order kinetic equation. These results are promising for future use of TCP for the remediation of polluted sites.
文摘Background Inflammation and immunity play a vital role in the pathogenesis of early brain injury after subarachnoid hemorrhage （SAH）. Nuclear factor-kappa B （NF-κB） regulates many genes essential for inflammation and immunity and is activated by toll-like receptor （TLR）. This study aimed to detect the expression of the toll-like receptor 4/nuclear factor-kappa B （TLR4/NF-κB） signaling in the rat brain after early SAH. Methods The rats were decapitated and their brains were removed at 0, 2, 4, 6, 12, 24 and 48 hours after a single injection of blood into the prechiasmatic cistern, mRNA expression of TLR4 was measured by Taqman real-time RT-PCR, and protein expression by immunohistochemistry and Western blotting. NF-κB activity and concentrations of tumor necrosis factor-alpha （TNF-α）, interleukin-lbeta （IL-1β） and interleukin-6 （IL-6） were measured by enzyme-linked immunosorbent assay （ELISA）. Results TaqMan real-time RT-PCR and Western blotting identified a biphasic change in TLR4 expression in both mRNA and protein： an initial peak （2-6 hours） and a sustained elevation （12-48 hours）. Immunohistochemical staining showed the inducible expression of TLR4-like immunoreactions predominantly in glial cells and vascular endothelium. A similar biphasic change in the activation of NF-κB subunit p65 as well as the production of NF-κB-regulated proinflammatory cytokines （TNF-α, IL-1β and IL-6） were detected by ELISA. Conclusions These data suggest that experimental SAH induces significant up-regulation of TLR4 expression and the NF-κB signaling in early brain injury. Activation of the TLR4/NF-κB signaling may regulate the inflammatory responses after SAH.
