针对风电全直流系统并网给交流电网带来的系统惯量降低、调频能力不足等问题,提出一种改善惯性响应与一次调频的变系数风电全直流系统协调控制策略。在惯量响应方面,网侧换流站采用惯性同步控制,直流升压站采用恒变比控制,实现直流电容...针对风电全直流系统并网给交流电网带来的系统惯量降低、调频能力不足等问题,提出一种改善惯性响应与一次调频的变系数风电全直流系统协调控制策略。在惯量响应方面,网侧换流站采用惯性同步控制,直流升压站采用恒变比控制,实现直流电容对电网的惯量支撑及直流低压侧的直流电压对交流系统频率的感知,在此基础上对直流风电机组(direct current wind turbine,DCWT)附加变虚拟惯性系数的虚拟惯量控制,使风电全直流系统在不同频率响应阶段具备不同的等效惯量。在一次调频方面,DCWT采用超速与变桨相结合的减载运行方式,通过变下垂控制来改变其有功出力,充分利用不同风速下的备用容量,使风电全直流系统更有效地参与一次调频。仿真算例表明,文中所提策略改善了风电全直流系统接入后电力系统的惯性响应及一次调频。展开更多
The legume–rhizobial symbiosis is a distinctive model in plant–microbe interactions.Despite being foreign invaders,rhizobia are acknowledged as“friends”by compatible host plants,leading to mutualistic endosymbiosi...The legume–rhizobial symbiosis is a distinctive model in plant–microbe interactions.Despite being foreign invaders,rhizobia are acknowledged as“friends”by compatible host plants,leading to mutualistic endosymbiosis(Cao et al.,2017).展开更多
Nodulation Receptor Kinase(NORK) functions as a co-receptor of Nod factor receptors to mediate rhizobial symbiosis in legumes, but its direct phosphorylation substrates that positively mediate root nodulation remain t...Nodulation Receptor Kinase(NORK) functions as a co-receptor of Nod factor receptors to mediate rhizobial symbiosis in legumes, but its direct phosphorylation substrates that positively mediate root nodulation remain to be fully identified.Here, we identified a GmNORK-Interacting Small Protein(GmNISP1) that functions as a phosphorylation target of GmNORK to promote soybean nodulation. GmNORKα directly interacted with and phosphorylated GmNISP1. Transcription of GmNISP1 was strongly induced after rhizobial infection in soybean roots and nodules. GmNISP1 encodes a peptide containing 90 amino acids with a “DY” consensus motif at its N-terminus.GmNISP1 protein was detected to be present in the apoplastic space. Phosphorylation of GmNISP1 by GmNORKα could enhance its secretion into the apoplast. Pretreatment with either purified GmNISP1 or phosphorylation-mimic GmNISP1~(12D) on the roots could significantly increase nodule numbers compared with the treatment with phosphorylation-inactive GmNISP1~(12A).The data suggested a model that soybean GmNORK phosphorylates GmNISP1 to promote its secretion into the apoplast, which might function as a potential peptide hormone to promote root nodulation.展开更多
文摘针对风电全直流系统并网给交流电网带来的系统惯量降低、调频能力不足等问题,提出一种改善惯性响应与一次调频的变系数风电全直流系统协调控制策略。在惯量响应方面,网侧换流站采用惯性同步控制,直流升压站采用恒变比控制,实现直流电容对电网的惯量支撑及直流低压侧的直流电压对交流系统频率的感知,在此基础上对直流风电机组(direct current wind turbine,DCWT)附加变虚拟惯性系数的虚拟惯量控制,使风电全直流系统在不同频率响应阶段具备不同的等效惯量。在一次调频方面,DCWT采用超速与变桨相结合的减载运行方式,通过变下垂控制来改变其有功出力,充分利用不同风速下的备用容量,使风电全直流系统更有效地参与一次调频。仿真算例表明,文中所提策略改善了风电全直流系统接入后电力系统的惯性响应及一次调频。
基金supported by the National Key R&D Program of China(2019YFA0904700)the National Natural Science Foundation of China(32090063)a Self-Innovation grant from the National Laboratory(AML2023B01)。
文摘The legume–rhizobial symbiosis is a distinctive model in plant–microbe interactions.Despite being foreign invaders,rhizobia are acknowledged as“friends”by compatible host plants,leading to mutualistic endosymbiosis(Cao et al.,2017).
基金supported by the National Natural Science Foundation of China (32090063 to Y.C.)the National Key R&D Program of China (2016YFD0100702 to Y.C.)+1 种基金the Natural Science Foundation of Hubei Province (2020CFA008 to Y.C.)a Fundamental Research Fund for the Central Universities (2662022SKYJ002 to Y.C.)。
文摘Nodulation Receptor Kinase(NORK) functions as a co-receptor of Nod factor receptors to mediate rhizobial symbiosis in legumes, but its direct phosphorylation substrates that positively mediate root nodulation remain to be fully identified.Here, we identified a GmNORK-Interacting Small Protein(GmNISP1) that functions as a phosphorylation target of GmNORK to promote soybean nodulation. GmNORKα directly interacted with and phosphorylated GmNISP1. Transcription of GmNISP1 was strongly induced after rhizobial infection in soybean roots and nodules. GmNISP1 encodes a peptide containing 90 amino acids with a “DY” consensus motif at its N-terminus.GmNISP1 protein was detected to be present in the apoplastic space. Phosphorylation of GmNISP1 by GmNORKα could enhance its secretion into the apoplast. Pretreatment with either purified GmNISP1 or phosphorylation-mimic GmNISP1~(12D) on the roots could significantly increase nodule numbers compared with the treatment with phosphorylation-inactive GmNISP1~(12A).The data suggested a model that soybean GmNORK phosphorylates GmNISP1 to promote its secretion into the apoplast, which might function as a potential peptide hormone to promote root nodulation.
