A sixty-one-year-old male,with a medical history of coronary heart disease and myocardial bridge (not confirmed by cardiac catheterization) in the past three years and a 1-h history of sudden onset nausea,retching,dia...A sixty-one-year-old male,with a medical history of coronary heart disease and myocardial bridge (not confirmed by cardiac catheterization) in the past three years and a 1-h history of sudden onset nausea,retching,diaphoresis,epigastric discomfort without any chest pain.The electrocardiogram revealed ST elevation myocardial infarction (STEMI) changes (Figure 1A).Urgent cardiac catheterization revealed mild myocardial bridge in the middle of the left anterior descending (LAD)(10% systolic compression) and normal left ventricular function without regional wall motion abnormalities.Three high-sensitivity cardiac troponin I (HSTNT) titers done at 4,8 and 20 h from the onset of symptoms were HSTNT =10.5 pg/mL,10.9 pg/mL,25.4 pg/mL (less than 26.2) with CK-MB = 3 pg/mL,18 pg/mL,13 pg/mL (< 25) and the myoglobin levels were = 34.8 pg/mL,39.6 pg/mL,44.5 pg/mL (< 140.1),respectively.展开更多
文摘A sixty-one-year-old male,with a medical history of coronary heart disease and myocardial bridge (not confirmed by cardiac catheterization) in the past three years and a 1-h history of sudden onset nausea,retching,diaphoresis,epigastric discomfort without any chest pain.The electrocardiogram revealed ST elevation myocardial infarction (STEMI) changes (Figure 1A).Urgent cardiac catheterization revealed mild myocardial bridge in the middle of the left anterior descending (LAD)(10% systolic compression) and normal left ventricular function without regional wall motion abnormalities.Three high-sensitivity cardiac troponin I (HSTNT) titers done at 4,8 and 20 h from the onset of symptoms were HSTNT =10.5 pg/mL,10.9 pg/mL,25.4 pg/mL (less than 26.2) with CK-MB = 3 pg/mL,18 pg/mL,13 pg/mL (< 25) and the myoglobin levels were = 34.8 pg/mL,39.6 pg/mL,44.5 pg/mL (< 140.1),respectively.
