Genuine Einstein-Podolsky-Rosen steering of generalized three-qubit states via unsharp measurements

We aim to explore all possible scenarios of(1→2)(where one wing is untrusted and the others two wings are trusted)and(2→1)(where two wings are untrusted,and one wing is trusted)genuine tripartite Einstein-Podolsky-Rosen(EPR)steering.The generalized Greenberger-Horne-Zeilinger(GHZ)state is shared between three spatially separated parties,Alice,Bob and Charlie.In both(1→2)and(2→1),we discuss the untrusted party and trusted party performing a sequence of unsharp measurements,respectively.For each scenario,we deduce an upper bound on the number of sequential observers who can demonstrate genuine EPR steering through the quantum violation of tripartite steering inequality.The results show that the maximum number of observers for the generalized GHZ states can be the same with that of the maximally GHZ state in a certain range of state parameters.Moreover,both the sharpness parameters range and the state parameters range in the scenario of(1→2)steering are larger than those in the scenario of(2→1)steering.

Reversible Data Hiding in Classification-Scrambling Encrypted-Image Based on Iterative Recovery

To improve the security and quality of decrypted images,this work proposes a reversible data hiding in encrypted image based on iterative recovery.The encrypted image is firstly generated by the pixel classification scrambling and bit-wise exclusive-OR(XOR),which improves the security of encrypted images.And then,a pixel-typemark generation method based on block-compression is designed to reduce the extra burden of key management and transfer.At last,an iterative recovery strategy is proposed to optimize the marked decrypted image,which allows the original image to be obtained only using the encryption key.The proposed reversible data hiding scheme in encrypted image is not vulnerable to the ciphertext-only attack due to the fact that the XOR-encrypted pixels are scrambled in the corresponding encrypted image.Experimental results demonstrate that the decrypted images obtained by the proposed method are the same as the original ones,and the maximum embedding rate of proposed method is higher than the previously reported reversible data hiding methods in encrypted image.

Genome-Wide Identification of the F-box Gene Family and Expression Analysis under Drought and Salt Stress in Barley

The F-box protein-encoding gene family plays an essential role in plant stress resistance.In present study,126 non-redundant F-box genes were identified in barley(Hordeum vulgare L.,Hv).The corresponding proteins contained 165–887 amino acid residues and all were amphiphilic,except 5 proteins.Phylogenetic analysis of F-box protein sequences in barley and stress-related F-box protein sequences in wheat and Arabidopsis thaliana(At)was used to classify barley F-box genes are divided into 9 subfamilies(A–I).A structure-based sequence alignment demonstrated that F-box proteins were highly conserved with a total of 10 conserved motifs.In total,124 F-box genes were unevenly distributed on 7 chromosomes;another 2 genes have not been anchored yet.The gene structure analysis revealed high variability in the number of exons and introns in F-box genes.Comprehensive analysis of expression profiles and phylogenetic tree analysis,a total of 12 F-box genes that may be related to stress tolerance in barley were screened.Of the 12 detected F-box genes,8 and 10 were upregulated after drought and salt stress treatments,respectively,using quantitative real-time polymerase chain reaction(qRT-PCR).This study is the first systematic analysis conducted on the F-box gene family in barley,which is of great importance for clarifying this family’s bioinformatic characteristics and elucidating its function in barley stress resistance.These results will serve as a theoretical reference for subsequent research on molecular regulation mechanisms,genetic breeding,and improvement.

Mapk7 deletion in chondrocytes causes vertebral defects by reducing MEF2C/PTEN/AKT signaling

Mutation of the MAPK7 gene was related to human scoliosis.Mapk7 regulated the development of limb bones and skulls in mice.However,the role of MAPK7 in vertebral development is still unclear.In this study,we constructed Col2a1-cre;Mapk7 f/f transgenic mouse model to delete Mapk7 in cartilage,which displayed kyphosis and osteopenia.Mechanistically,Mapk7 loss decreased MEF2C expression and thus activated PTEN to oppose PI3K/AKT signaling in vertebral growth plate chondrocytes,which impaired chondrocyte hypertrophy and attenuated vertebral ossification.In vivo,systemic pharmacological activation of AKT rescued impaired chondrocyte hypertrophy and alleviated mouse vertebral defects caused by Mapk7 deficiency.Our study firstly clarified the mechanism by which MAPK7 was involved in vertebral development,which might contribute to understanding the pathology of spinal deformity and provide a basis for the treatment of developmental disorders of the spine.