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DLX3(Q178R)mutation delays osteogenic differentiation via H19/miR-29c-3p/KDM5B axis in TDO-iPSCs-derived MSCs
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作者 Liying Dong Na Zhao +6 位作者 Dongmei Wang Meng Wang Yixin Zhang Liangjie Sun Chong Ding Yixiang Wang zeyun ma 《Genes & Diseases》 SCIE 2024年第4期49-52,共4页
Tricho-dento-osseous(TDO)syndrome is a rare autosomal dominant disease resulting from distal-less homeobox 3(DLX3)mutation.1,2 Accumulative bone density in alveolar bone is a clinically favorable phenotype for TDO pat... Tricho-dento-osseous(TDO)syndrome is a rare autosomal dominant disease resulting from distal-less homeobox 3(DLX3)mutation.1,2 Accumulative bone density in alveolar bone is a clinically favorable phenotype for TDO patients.However,the limited number of bone marrow mesenchymal stem cells(BMSCs)in TDO patients restricts their application. 展开更多
关键词 patients clinically alveolar
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