Deep learning has emerged in many practical applications,such as image classification,fault diagnosis,and object detection.More recently,convolutional neural networks(CNNs),representative models of deep learning,have ...Deep learning has emerged in many practical applications,such as image classification,fault diagnosis,and object detection.More recently,convolutional neural networks(CNNs),representative models of deep learning,have been used to solve fault detection.However,the current design of CNNs for fault detection of wind turbine blades is highly dependent on domain knowledge and requires a large amount of trial and error.For this reason,an evolutionary YOLOv8 network has been developed to automatically find the network architecture for wind turbine blade-based fault detection.YOLOv8 is a CNN-backed object detection model.Specifically,to reduce the parameter count,we first design an improved FasterNet module based on the Partial Convolution(PConv)operator.Then,to enhance convergence performance,we improve the loss function based on the efficient complete intersection over the union.Based on this,a flexible variable-length encoding is proposed,and the corresponding reproduction operators are designed.Related experimental results confirmthat the proposed approach can achieve better fault detection results and improve by 2.6%in mean precision at 50(mAP50)compared to the existing methods.Additionally,compared to training with the YOLOv8n model,the YOLOBFE model reduces the training parameters by 933,937 and decreases the GFLOPS(Giga Floating Point Operations Per Second)by 1.1.展开更多
针对无线射频识别(radio frequency identification,RFID)系统中的标签碰撞问题,在预先侦测四元查询树算法的基础上,提出一种改进的预先侦测查询树防碰撞算法。结合后退式搜索与八叉树询问机制,通过提取碰撞位信息来构建查询树。实验结...针对无线射频识别(radio frequency identification,RFID)系统中的标签碰撞问题,在预先侦测四元查询树算法的基础上,提出一种改进的预先侦测查询树防碰撞算法。结合后退式搜索与八叉树询问机制,通过提取碰撞位信息来构建查询树。实验结果表明,改进的算法在通信量、碰撞时期的标签数量、阅读器的查询次数以及系统的吞吐率方面都优于BBS、PDQT算法。展开更多
OBJECTIVE ： The pathogenesis of tardive dyskinesia （TD） is complicated and uncertain, Thus, there is not any effective treatment for it. The psychiatrists pay more and more attention to TD, which lasts for a long t...OBJECTIVE ： The pathogenesis of tardive dyskinesia （TD） is complicated and uncertain, Thus, there is not any effective treatment for it. The psychiatrists pay more and more attention to TD, which lasts for a long time and is difficult to treat. DATA SOURCES： A computer-based online search of Medline database was undertaken to identify articles about the feature of etiology and the progression of treatment for TD published in English by using the keywords of ＂rD, etiology, pathogenesis＂ and ＂TD, therapy, drug treatment＂. Meanwhile, Chinese articles about the feature of etiology and the progression of treatment for TD were searched in Wanfang database and China journal full-text database, and the keywords were ＂TD, etiology, pathogenesis＂ and ＂TD, therapy, drug Treatment＂ in Chinese. STUDY SELECTION： Articles met the following inclusion criteria were selected in this paper. Inclusion criteria： （1） Researches of randomized blind control design, before and after control design and retrospective. （2） Researches of the feature of etiology and the progression of treatment for TD. Exclusion criteria： the repetitive researches and individual reports. DATA EXTRACTION ： Totally 65 articles related the feature of etiology and the progression of treatment for TD of randomized blind control design, before and after control design and retrospective studies were collected, and 53 of them were accorded with the inclusion criteria. Of the 12 excluded ones, 8 were concerning with genetics, 4 were repetitive researches. DATA SYNTHESIS ： The feature of etiology for TD includes：（1） Hypothesis of dopamine receptor super-sensitivity： The dopamine receptor is persistently blocked, so it will result in functional disorder in CNS, and then TD may take place. （2）） Hypothesis of neuronal degeneration： The concentration of aminosuccinic acid and glutamic acid will increase after the antipsychotic used for a long time and this will result in neuronal degeneration through glutamic acid receptor in the postsynaptic membrane; meanwhile with free radical, the nerve cells of corpus striatum may degenerate and become necrosis. （3） Sex and age： The females and gerontal patients are liability to the TD disease. It is may related to the lower estrogen. （4） Molecule heredity： TD may association with the dopamine and 5-HT receptor gene polymorphism. （5） Other theories： Hypofunction of γ-amino-butyri acid （GABA）, hypothesis of noradrenaline 5-serotonin and nutrition metabolism can cause TD disease. Treatlent for TD： （1） Dopamine receptor agonist： The therapeutic effect is not satisfactory, especially for gerontism females. （2） Oxygen free radical scavenger： As represent of vitamin E, it can clear out free radicals and reduce the potential cytotoxic effect of free radicals. （3） Calcium channel blocker： This maybe related to block calcium ions releasing from muscle cells and inhibit muscle convulsion; therefore, it can be used for symptomatic treatment. （4） GABA receptor agonist： It is more effective for the prominent dysmyotonia than dancing slowly symptom. （5） Antipsychotic： There is some therapeutic effect with ciozapine, but the effect will reduce because of the age growing up and the symptom exacerbating. （6） Other therapies： Valproate sodium, cyproheptadine, melatonin, branched chain amino acid, ahalysantinfarctasum, electric acupuncture and injection ad acumen, traditional Chinese drug have a certain effects on TD. Prevention of TD： The serum creatine phosphokinase （CPK） combined with symptoms should be checked regularly so as to early discovery TD. CONCLUSION ： （1） Etiology of TD： The hypothesis of dopamine receptor super-sensitivity is denyed; the hypothesis of neuronal degeneration is approved in academic circles; the sex and age is a finding of generally received; but the dopamine and 5-HT receptor gene polymorphism, hypofunction of GABA, noradrenaline, 5-serotonin and nutrition metabolism cannot explain the pathogenesis of TD. （2） Treatment for TD： The therapeutic effect of dopamine receptor agonist is not satisfactory; the oxygen free radical scavenger maybe effective; calcium channel blocker maybe used for symptomatic treatment; GABA receptor agonist maybe more effective for the prominent dysmyotonia than dancing slowly symptom; the consequence of antipsychotic is discrepancy; other therapies maybe use to adjunctive therapies. （3） As far as prevention of TD is concerned, and the serum CPK combined with symptoms should be checked regularly so as to early discovery TD.展开更多
基金supported by the Liaoning Province Applied Basic Research Program Project of China(Grant:2023JH2/101300065)the Liaoning Province Science and Technology Plan Joint Fund(2023-MSLH-221).
文摘Deep learning has emerged in many practical applications,such as image classification,fault diagnosis,and object detection.More recently,convolutional neural networks(CNNs),representative models of deep learning,have been used to solve fault detection.However,the current design of CNNs for fault detection of wind turbine blades is highly dependent on domain knowledge and requires a large amount of trial and error.For this reason,an evolutionary YOLOv8 network has been developed to automatically find the network architecture for wind turbine blade-based fault detection.YOLOv8 is a CNN-backed object detection model.Specifically,to reduce the parameter count,we first design an improved FasterNet module based on the Partial Convolution(PConv)operator.Then,to enhance convergence performance,we improve the loss function based on the efficient complete intersection over the union.Based on this,a flexible variable-length encoding is proposed,and the corresponding reproduction operators are designed.Related experimental results confirmthat the proposed approach can achieve better fault detection results and improve by 2.6%in mean precision at 50(mAP50)compared to the existing methods.Additionally,compared to training with the YOLOv8n model,the YOLOBFE model reduces the training parameters by 933,937 and decreases the GFLOPS(Giga Floating Point Operations Per Second)by 1.1.
文摘针对无线射频识别(radio frequency identification,RFID)系统中的标签碰撞问题,在预先侦测四元查询树算法的基础上,提出一种改进的预先侦测查询树防碰撞算法。结合后退式搜索与八叉树询问机制,通过提取碰撞位信息来构建查询树。实验结果表明,改进的算法在通信量、碰撞时期的标签数量、阅读器的查询次数以及系统的吞吐率方面都优于BBS、PDQT算法。
文摘OBJECTIVE ： The pathogenesis of tardive dyskinesia （TD） is complicated and uncertain, Thus, there is not any effective treatment for it. The psychiatrists pay more and more attention to TD, which lasts for a long time and is difficult to treat. DATA SOURCES： A computer-based online search of Medline database was undertaken to identify articles about the feature of etiology and the progression of treatment for TD published in English by using the keywords of ＂rD, etiology, pathogenesis＂ and ＂TD, therapy, drug treatment＂. Meanwhile, Chinese articles about the feature of etiology and the progression of treatment for TD were searched in Wanfang database and China journal full-text database, and the keywords were ＂TD, etiology, pathogenesis＂ and ＂TD, therapy, drug Treatment＂ in Chinese. STUDY SELECTION： Articles met the following inclusion criteria were selected in this paper. Inclusion criteria： （1） Researches of randomized blind control design, before and after control design and retrospective. （2） Researches of the feature of etiology and the progression of treatment for TD. Exclusion criteria： the repetitive researches and individual reports. DATA EXTRACTION ： Totally 65 articles related the feature of etiology and the progression of treatment for TD of randomized blind control design, before and after control design and retrospective studies were collected, and 53 of them were accorded with the inclusion criteria. Of the 12 excluded ones, 8 were concerning with genetics, 4 were repetitive researches. DATA SYNTHESIS ： The feature of etiology for TD includes：（1） Hypothesis of dopamine receptor super-sensitivity： The dopamine receptor is persistently blocked, so it will result in functional disorder in CNS, and then TD may take place. （2）） Hypothesis of neuronal degeneration： The concentration of aminosuccinic acid and glutamic acid will increase after the antipsychotic used for a long time and this will result in neuronal degeneration through glutamic acid receptor in the postsynaptic membrane; meanwhile with free radical, the nerve cells of corpus striatum may degenerate and become necrosis. （3） Sex and age： The females and gerontal patients are liability to the TD disease. It is may related to the lower estrogen. （4） Molecule heredity： TD may association with the dopamine and 5-HT receptor gene polymorphism. （5） Other theories： Hypofunction of γ-amino-butyri acid （GABA）, hypothesis of noradrenaline 5-serotonin and nutrition metabolism can cause TD disease. Treatlent for TD： （1） Dopamine receptor agonist： The therapeutic effect is not satisfactory, especially for gerontism females. （2） Oxygen free radical scavenger： As represent of vitamin E, it can clear out free radicals and reduce the potential cytotoxic effect of free radicals. （3） Calcium channel blocker： This maybe related to block calcium ions releasing from muscle cells and inhibit muscle convulsion; therefore, it can be used for symptomatic treatment. （4） GABA receptor agonist： It is more effective for the prominent dysmyotonia than dancing slowly symptom. （5） Antipsychotic： There is some therapeutic effect with ciozapine, but the effect will reduce because of the age growing up and the symptom exacerbating. （6） Other therapies： Valproate sodium, cyproheptadine, melatonin, branched chain amino acid, ahalysantinfarctasum, electric acupuncture and injection ad acumen, traditional Chinese drug have a certain effects on TD. Prevention of TD： The serum creatine phosphokinase （CPK） combined with symptoms should be checked regularly so as to early discovery TD. CONCLUSION ： （1） Etiology of TD： The hypothesis of dopamine receptor super-sensitivity is denyed; the hypothesis of neuronal degeneration is approved in academic circles; the sex and age is a finding of generally received; but the dopamine and 5-HT receptor gene polymorphism, hypofunction of GABA, noradrenaline, 5-serotonin and nutrition metabolism cannot explain the pathogenesis of TD. （2） Treatment for TD： The therapeutic effect of dopamine receptor agonist is not satisfactory; the oxygen free radical scavenger maybe effective; calcium channel blocker maybe used for symptomatic treatment; GABA receptor agonist maybe more effective for the prominent dysmyotonia than dancing slowly symptom; the consequence of antipsychotic is discrepancy; other therapies maybe use to adjunctive therapies. （3） As far as prevention of TD is concerned, and the serum CPK combined with symptoms should be checked regularly so as to early discovery TD.
