Research on Decalcification Degradation Process of Cement Stone

An accelerated laboratory method（saturated ammonium nitrate solution immersion method） was used to analyze the degradation of cement decalcification process. By studying the changes of intensity, volume, elastic modulus, quality, p H value, the Ca/Si, and mineral phase, it could be found that the first cement decalcification degradation process was the decalcification of calcium hydroxide, and then CSH gel, AFm, etc. The secondary ettringite deposition happened and the decalcification degradation depth was proportional to the square root of time. Moreover, the corresponding strength of cement would be gradually reduced, cement rock volume shrinkage occurred, p H values decreased, the surface elastic modulus decreased down to a certain level, and slightly changed and the Ca/Si was 3.1 from the beginning and lasted down to 1.3.

Characteristics of Hydrotreating Reaction in VRDS Units Located along the Yangtze River and Overall Solution for Long-cycle Running

The VRDS feedstock processed in refineries along the Yangtze River has special characteristics, including high Fe and Ca content, and low sulfur and high nitrogen content. Depending upon feedstock properties and operating conditions,some approaches have been developed by the SINOPEC Research Institute of Petroleum Processing(RIPP), which include installing the decalcification facility, developing new guard catalysts and HDCCR catalysts, implementing a new catalyst grading approach, developing a highly efficient distribution technology and applying RICP process in some refineries.The application effects have revealed that the integrated technology, which can be conducive to the long-cycle operation developed by RIPP, can maximize the deposits uptake capacity of the guard reactor and the activity of grading catalysts.

Standardization of experimental animals temporal bone sections

Preparation of the temporal bone for light microscopy is an important step in histological studies of the inner ear. Due to the complexity of structures of the inner ear, it is difficult to measure or compare structures of interest without a commonly accepted standardized measure of temporal bone sections. Therefore, standardization of temporal bone sections is very important for histological assessment of sensory hair cells and peripheral ganglion neurons in the cochlear and vestibular systems. The standardized temporal bone sectioning is oriented to a plane parallel to the outer and internal auditory canals. Sections are collected from the epitympanum to the hypotympanum to reveal layers in the order of the crista ampullaris of the superior and lateral semicircular canals, macula utriculi and macula sacculi, superior vestibular ganglion neurons, macula of saccule and inferior vestibular ganglion neurons, cochlear modiolus, endolymphatic duct and endolymphatic sac, and finally the crista ampullaris of the posterior semicircular canal. Moreover, technical details of preparing for temporal bone sectioning including fixation, decalcification, whole temporal bone staining, embedding penetration, and embedding orientation are also discussed.

A Review of Nutrients to Extend Healthspan and Avoid Cancer by Reducing the Amount of Protein Misfolding, Free Radicals, and Calcification

Two major causes of human aging include protein misfolding and free radicals. Protein misfolding occurs when proteins which are synthesized by cells do not have the proper amino acid sequence or do not achieve the correct three-dimensional configuration to function properly. Peer-reviewed scientific literature explains how these processes contribute to many age-associated diseases. A few examples include cancer, heart disease, dementias including Parkinson’s and Alzheimer’s diseases, and arthritis. This article reviews how protein misfolding can be slowed and even reversed by appropriate nutrition, potentially slowing and reversing these diseases. One cause of misfolding is mRNA translation occurring too rapidly for proper chaperone binding or protein folding. A second cause is deficiency of amino acids so improper tRNA binding occurs. A third cause is free radicals. They cause mutations promoting misfolding and cancer, and oxidize lipoproteins causing plaque in circulation promoting heart disease and stroke. Nutrients with proven actions will contribute to longer healthspans for our aging population. Healthspan is the number of healthy years before chronic or terminal diseases substantially impair the quality of life. This can be done especially by slowing and reversing these three causes of PM. Niacin, quercetin, EGCG, alpha-lipoic acid, N-acetyl-carnitine, tyrosine and cysteine address protein misfolding. Vitamin C and glutathione trap free radicals. Vitamin K amplifies free radical cancer killing by vitamin C and activates decalcification enzymes which remove calcium deposits in the circulatory system and strengthen bones. Apigenin activates the pathway of caloric restriction and induces cancer cell apoptosis. This article provides citations and explanations of the progress showing new ways to maintain health as we age. For convenience and cost savings, many of these ingredients can be consumed in supplement form, taken twice a day to maintain water-soluble nutrient levels.

Genetic characteristics and molecular diagnostics of bone tumors

The diagnosis of primary tumors of bone relies heavily on clinicopathological and radiological correlation and is often best performed in a multidisciplinary setting.Bone tumors comprise a heterogenous category of human lesions ranging from benign to malignant neoplasms.These tumors affect a wide age range and can become problematic for diagnosis when less common entities are encountered.Traditionally the pathological diagnosis of many bone tumors has been based primarily on the evaluation of hematoxylin and eosin-stained glass slides,sometimes combined with ancillary diagnostic techniques such as immunohistochemistry,conventional cytogenetics,fluorescence in situ hybridization,and polymerase chain reaction-based assays.More recently,the advent of massively parallel sequencing-based techniques has opened new avenues for diagnostic testing in bone tumors;however,these new testing modalities are sensitive to traditional decalcification procedures that are commonly used in the routine processing of bony specimens.Herein we provide a focused review concentrating on the molecular genetic features of bone tumors with specific,recurrent genetic alterations that make them appealing targets for directed ancillary testing by conventional or molecular techniques.In addition,specimen handling with regards to decalcification procedures are discussed and the different types of testing modalities available are reviewed.