介绍一种L波段单级高线性低噪声放大器的工作原理和设计方法。与传统的接收机射频前端放大器主要考虑低噪声和高增益特性不同,文中选用了低成本、低功耗的S iGe NPN B JT器件设计高三阶交截点的低噪声放大器。设计中利用了微波CAD工具...介绍一种L波段单级高线性低噪声放大器的工作原理和设计方法。与传统的接收机射频前端放大器主要考虑低噪声和高增益特性不同,文中选用了低成本、低功耗的S iGe NPN B JT器件设计高三阶交截点的低噪声放大器。设计中利用了微波CAD工具对电路进行仿真与优化,同时对生成的微带印刷电路板进行了电磁仿真。展开更多
In the present study, the radioreceptor binding method was used to determine the changes of IP3 content in rat brain and dorsal spinal cord of high frequency (100 Hz) electroacupuncture (EA) analgesia and EA tolerance...In the present study, the radioreceptor binding method was used to determine the changes of IP3 content in rat brain and dorsal spinal cord of high frequency (100 Hz) electroacupuncture (EA) analgesia and EA tolerance rat. The control levels of IP3 in rat brain (less cerebellum and cortex) and dorsal spinal cord were 6.3± 0.78 pmol / mg protein and 3.4± 0.60 pmol / mg protein, respectively. The results showed that IP, in brain increased gradually within 45 min after stimulation in EA analgesia rat. Meanwhile, the dorsal spinal cord IP, content decreased significantly 15 min, 30 min after EA stimulation and recovered to control level 45 min after EA stimulation. In EA tolerance rat, IP3 contents markedly increased in brain. And IP3 content in the spinal cord also increased dramatically within 30 min, but decreased rapidly to control level 45 min after EA stimulation. The IP3 level in EA tolerance rat brain and spinal cord was much higher than that in EA analgesia rat (P<0.01). The studies first reported that high frequency (100 Hz) EA may be linked to PI system in its signal transduction pathways.展开更多
文摘介绍一种L波段单级高线性低噪声放大器的工作原理和设计方法。与传统的接收机射频前端放大器主要考虑低噪声和高增益特性不同,文中选用了低成本、低功耗的S iGe NPN B JT器件设计高三阶交截点的低噪声放大器。设计中利用了微波CAD工具对电路进行仿真与优化,同时对生成的微带印刷电路板进行了电磁仿真。
文摘In the present study, the radioreceptor binding method was used to determine the changes of IP3 content in rat brain and dorsal spinal cord of high frequency (100 Hz) electroacupuncture (EA) analgesia and EA tolerance rat. The control levels of IP3 in rat brain (less cerebellum and cortex) and dorsal spinal cord were 6.3± 0.78 pmol / mg protein and 3.4± 0.60 pmol / mg protein, respectively. The results showed that IP, in brain increased gradually within 45 min after stimulation in EA analgesia rat. Meanwhile, the dorsal spinal cord IP, content decreased significantly 15 min, 30 min after EA stimulation and recovered to control level 45 min after EA stimulation. In EA tolerance rat, IP3 contents markedly increased in brain. And IP3 content in the spinal cord also increased dramatically within 30 min, but decreased rapidly to control level 45 min after EA stimulation. The IP3 level in EA tolerance rat brain and spinal cord was much higher than that in EA analgesia rat (P<0.01). The studies first reported that high frequency (100 Hz) EA may be linked to PI system in its signal transduction pathways.
