英国公司法中的Foss v. Harbottle规则及其例外是英国公司法保护少数派股东权利的一种独特的救济方式。它不仅影响了英美法系国家的股东代表诉讼制度,而且影响着部分大陆法系国家和地区的股东代表诉讼制度。尽管中国新公司法已经确定了...英国公司法中的Foss v. Harbottle规则及其例外是英国公司法保护少数派股东权利的一种独特的救济方式。它不仅影响了英美法系国家的股东代表诉讼制度,而且影响着部分大陆法系国家和地区的股东代表诉讼制度。尽管中国新公司法已经确定了自己的股东代表制度,但英国在代表诉讼制度上的保守态度和经验,依然值得我们借鉴。展开更多
Intense exercise may cause heart remodeling to compensate increases in blood pressure or volume by increasing muscle mass. Cardiac changes do not involve only the left ventricle, but all heart chambers. Physiological ...Intense exercise may cause heart remodeling to compensate increases in blood pressure or volume by increasing muscle mass. Cardiac changes do not involve only the left ventricle, but all heart chambers. Physiological cardiac modeling in athletes is associated with normal or enhanced cardiac function, but recent studies have documented decrements in left ventricular function during intense exercise and the release of cardiac markers of necrosis in athlete's blood of uncertain significance. Furthermore, cardiac remodeling may predispose athletes to heart disease and result in electrical remodeling, responsible for arrhythmias. Athlete's heart is a physiological condition and does not require a specific treatment. In some conditions, it is important to differentiate the physiological adaptations from pathological conditions, such as hypertrophic cardiomyopathy, arrhythmogenic dysplasia of the right ventricle, and non-compaction myocardium, for the greater risk of sudden cardiac death of these conditions. Moreover, some drugs and performance-enhancing drugs can cause structural alterations and arrhythmias, therefore, their use should be excluded.展开更多
文摘英国公司法中的Foss v. Harbottle规则及其例外是英国公司法保护少数派股东权利的一种独特的救济方式。它不仅影响了英美法系国家的股东代表诉讼制度,而且影响着部分大陆法系国家和地区的股东代表诉讼制度。尽管中国新公司法已经确定了自己的股东代表制度,但英国在代表诉讼制度上的保守态度和经验,依然值得我们借鉴。
文摘Intense exercise may cause heart remodeling to compensate increases in blood pressure or volume by increasing muscle mass. Cardiac changes do not involve only the left ventricle, but all heart chambers. Physiological cardiac modeling in athletes is associated with normal or enhanced cardiac function, but recent studies have documented decrements in left ventricular function during intense exercise and the release of cardiac markers of necrosis in athlete's blood of uncertain significance. Furthermore, cardiac remodeling may predispose athletes to heart disease and result in electrical remodeling, responsible for arrhythmias. Athlete's heart is a physiological condition and does not require a specific treatment. In some conditions, it is important to differentiate the physiological adaptations from pathological conditions, such as hypertrophic cardiomyopathy, arrhythmogenic dysplasia of the right ventricle, and non-compaction myocardium, for the greater risk of sudden cardiac death of these conditions. Moreover, some drugs and performance-enhancing drugs can cause structural alterations and arrhythmias, therefore, their use should be excluded.