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后奥斯维辛的梦魇叙事——论马内阿长篇小说《黑信封》中的“创伤记忆” 被引量:1
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作者 陈恒仕 《苏州大学学报(哲学社会科学版)》 CSSCI 北大核心 2010年第6期132-135,共4页
本文试图考察的是马内阿的长篇小说《黑信封》中叙事特征,及其内化于叙事内部的批判力量,着力分析小说以书写极权时代"创伤记忆"为特征的"梦魇叙事"在文本中的具体展开形式,正是这种文本内部"现实与梦幻的交织... 本文试图考察的是马内阿的长篇小说《黑信封》中叙事特征,及其内化于叙事内部的批判力量,着力分析小说以书写极权时代"创伤记忆"为特征的"梦魇叙事"在文本中的具体展开形式,正是这种文本内部"现实与梦幻的交织"的形式力量,实现了对外部社会的否定和批判。 展开更多
关键词 后奥斯维辛 “创伤记忆” 梦魇叙事 极权主义
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《爵士乐》中的“创伤重演”和“创伤消解”
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作者 唐小霞 《西南科技大学学报(哲学社会科学版)》 2015年第1期42-47,共6页
从创伤理论的视角来看,《爵士乐》中非裔美国人的内心无不潜藏着带有种族迫害印痕的、难以言说的个体和集体创伤记忆,而创伤的延宕性和强迫重复性,使得这些受创者在后奴隶制年代仍然深受"创伤重演"的折磨。小说通过相互叙述... 从创伤理论的视角来看,《爵士乐》中非裔美国人的内心无不潜藏着带有种族迫害印痕的、难以言说的个体和集体创伤记忆,而创伤的延宕性和强迫重复性,使得这些受创者在后奴隶制年代仍然深受"创伤重演"的折磨。小说通过相互叙述与见证,将"创伤记忆"转变为"叙述记忆"黑人群体回忆并重构了种族创伤史,最终实现了"创伤消解"和黑人社区凝聚力的恢复。 展开更多
关键词 《爵士乐》 “创伤重演” “创伤消解” “创伤记忆” “叙述记忆”
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NMDA receptor antagonist MK-801 reduces neuronal damage and preserves learning and memory in a rat model of traumatic brain injury 被引量:3
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作者 韩瑞璋 胡金家 +2 位作者 翁原驰 李丁峰 黄艺 《Neuroscience Bulletin》 SCIE CAS CSCD 2009年第6期367-375,共9页
Objective NMDA receptor channel plays an important role in the pathophysiological process of traumatic brain injury (TBI). The present study aims to study the pathological mechanism of TBI and the impairment of lear... Objective NMDA receptor channel plays an important role in the pathophysiological process of traumatic brain injury (TBI). The present study aims to study the pathological mechanism of TBI and the impairment of learning and memory after TBI, and to investigate the mechanism of the protective effect of NMDA receptor antagonist MK-801 on learning and memory disorder after TBI. Methods Forty Sprague-Dawley rats (weighing approximately 200 g) were randomized into 5 groups (n = 8 in each group): control group, model group, low-dose group (MK-801 0.5 mg/kg), middle-dose group (MK-801 2 mg/kg), and high-dose group (MK-801 10 mg/kg). TBI model was established using a weight-drop head injury mode. After 2-month drug treatment, learning and memory ability was evaluated by using Morris water maze test. Then the animals were sacrificed, and brain tissues were taken out for morphological and immunohistochemical assays. Results The ability of learning and memory was significantly impaired in the TBI model animals. Besides, the neuronal caspase-3 expression, neuronal nitric oxide synthase (nNOS)-positive neurons and OX-42-positive microglia were all increased in TBI animals. Meanwhile, the number of neuron synapses was decreased, and vacuoles degeneration could be observed in mitochondria. After MK-801 treatment at 3 different dosages, the ability of learning and memory was markedly improved, as compared to that of the TBI model animals. Moreover, neuronal caspase-3 expression, OX-42-positive microglia and nNOS-positive neurons were all significantly decreased. Meanwhile, the mitochondria degeneration was greatly inhibited. Conclusion MK-801 could significantly inhibit the degeneration and apoptosis of neurons in damaged brain areas. It could also inhibit TBI-induced increase in nNOS-positive neurons and OX-42-positive microglia. Impairment in learning and memory in TBI animals could be repaired by treatment with MK-801. 展开更多
关键词 traumatic brain injury MK-801 learning and memory CASPASE-3 MICROGLIA neuronal nitric oxide synthase
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