利用黑龙江省2012年16 d 250 m的MODIS NDVI数据,使用Savitsky-Golay滤波法、Logistic调合函数模型和非对称Gaussian模型3种方法对曲线拟合重构,引入均方根误差比较3种方法的优劣;在曲线平滑的基础上提取物候参数,分别对森林和农用地用...利用黑龙江省2012年16 d 250 m的MODIS NDVI数据,使用Savitsky-Golay滤波法、Logistic调合函数模型和非对称Gaussian模型3种方法对曲线拟合重构,引入均方根误差比较3种方法的优劣;在曲线平滑的基础上提取物候参数,分别对森林和农用地用固定阈值和动态阈值提取其物候参数,目的在于比较两种阈值设定方法的优劣,并获取阈值使物候参数结果与作物农事历信息吻合。结果表明,Savitsky-Golay滤波法在与原始曲线近似度方面优于另外两种拟合方法。展开更多
Objective: This study aims to investigate the effects of urocortin (Ucn) on the viability of endothelial cells (ECV304) and rat vascular muscle cells (VSMC). Methods: Rat aortic VSMC were isolated from the rats' t...Objective: This study aims to investigate the effects of urocortin (Ucn) on the viability of endothelial cells (ECV304) and rat vascular muscle cells (VSMC). Methods: Rat aortic VSMC were isolated from the rats' thoracic aorta. We studied the effect of Ucn on the viability of ECV304 cells and VSMC by using a tetrazolium (MTT) assay.Results: Ucn (10 -7 mol/L) inhibited the viability of ECV304 cells and VSMC. Inhibition rates are 13% and 15%, respectively(P<0.05, compared with Control). This inhibition was not dependent on the affecting time and was not affected by the addition of ATP-sensitive potassium channel (KATP channel) blocker, glybenclamide (Gly, 10 mol/L). Conclusion: Ucn inhibits the viability of ECV304 and VSMC. Our results suggest that Ucn may be a new vasoactive agent and may have a beneficial effect in the process of vascular remodeling (VR).展开更多
文摘利用黑龙江省2012年16 d 250 m的MODIS NDVI数据,使用Savitsky-Golay滤波法、Logistic调合函数模型和非对称Gaussian模型3种方法对曲线拟合重构,引入均方根误差比较3种方法的优劣;在曲线平滑的基础上提取物候参数,分别对森林和农用地用固定阈值和动态阈值提取其物候参数,目的在于比较两种阈值设定方法的优劣,并获取阈值使物候参数结果与作物农事历信息吻合。结果表明,Savitsky-Golay滤波法在与原始曲线近似度方面优于另外两种拟合方法。
文摘Objective: This study aims to investigate the effects of urocortin (Ucn) on the viability of endothelial cells (ECV304) and rat vascular muscle cells (VSMC). Methods: Rat aortic VSMC were isolated from the rats' thoracic aorta. We studied the effect of Ucn on the viability of ECV304 cells and VSMC by using a tetrazolium (MTT) assay.Results: Ucn (10 -7 mol/L) inhibited the viability of ECV304 cells and VSMC. Inhibition rates are 13% and 15%, respectively(P<0.05, compared with Control). This inhibition was not dependent on the affecting time and was not affected by the addition of ATP-sensitive potassium channel (KATP channel) blocker, glybenclamide (Gly, 10 mol/L). Conclusion: Ucn inhibits the viability of ECV304 and VSMC. Our results suggest that Ucn may be a new vasoactive agent and may have a beneficial effect in the process of vascular remodeling (VR).