目的:探讨糖尿病患者性激素的改变与糖尿病性ED的关系。方法:将80例糖尿病的男性患者,根据其有无ED分为糖尿病ED组与糖尿病非ED组,所有患者均排除了同时有肝、肾、甲状腺疾病,并停用了各种影响勃起功能和性激素的药物,应用放射免疫分析...目的:探讨糖尿病患者性激素的改变与糖尿病性ED的关系。方法:将80例糖尿病的男性患者,根据其有无ED分为糖尿病ED组与糖尿病非ED组,所有患者均排除了同时有肝、肾、甲状腺疾病,并停用了各种影响勃起功能和性激素的药物,应用放射免疫分析法(RIA)检测分两组患者的血清睾酮(T)、催乳素(PRL)和雌二醇(E2)水平,并比较两组患者的临床资料。结果:糖尿病ED组睾酮明显低于非ED组(19.5±5.2ng/L vs 90±7.5ng/L),两组的PRL、E2、血糖无显著差异,分别为(12.5±2.2μg/L vs 12.7±2.1μg/L)、(36.56±0.53ng/L vs 37.24±0.54ng/L)、(10.2±3.6mmo1/L vs 9.8±3.4mmo1/L)。ED组患者年龄明显高于非ED组(52.4±8.1岁vs 42.4±5.4岁)。两组患者糖尿病病程分别为33.4±10.6月vs 10.9±4.1月,两组相比有显著差异。结论:糖尿病致睾酮的下降是糖尿病性ED产生的原因之一,患者的年龄及糖尿病病程也与糖尿病性ED相关。展开更多
In this article, we have reviewed the hepatic perfusion disorder （HPD）, etiopathogenesis of HPD and corresponding diseases. Review of the literature was based on computer searches （PubMed, Index Medicus） and perso...In this article, we have reviewed the hepatic perfusion disorder （HPD）, etiopathogenesis of HPD and corresponding diseases. Review of the literature was based on computer searches （PubMed, Index Medicus） and personal experiences. We considered HPD reflects perfusion differences due to redistribution of arterial blood flow among segments, subsegments and lobes of the liver. The plain CT scan findings of HPD manifests as triangular or wedge-shaped areas of low attenuation. On contrast-enhanced CT scan, HPD manifests multiple （or single） transient wedge-shaped, rotundloid or irregular appearance, homogeneous hyperattenuation （in less cases, hypoattenuation） during the hepatic arterial phase （HAP） and isoattenuated or slightly hyperattenuated areas during the portal arterial phase. Dynamic enhanced magnetic resonance （MR） features are similar to enhanced CT scan. Angiographic findings include non-opacification of portal vein on portograms or wedge-shaped segmental staining in arterial and parenchymal phases on hepatic angiograms. The causes of HPD are arterioportal shunts （APS）, intrahepatic vascular compressions and portal vein occlusion, steal phenomenon by hypervascular tumors, vascular variations and any other unknown reasons. It is very important for radiologists to be familiar with the various appearances of HPD to avoid false-positive diagnosis of pseudolesions and not to overestimate the extent of the disease.展开更多
文摘目的:探讨糖尿病患者性激素的改变与糖尿病性ED的关系。方法:将80例糖尿病的男性患者,根据其有无ED分为糖尿病ED组与糖尿病非ED组,所有患者均排除了同时有肝、肾、甲状腺疾病,并停用了各种影响勃起功能和性激素的药物,应用放射免疫分析法(RIA)检测分两组患者的血清睾酮(T)、催乳素(PRL)和雌二醇(E2)水平,并比较两组患者的临床资料。结果:糖尿病ED组睾酮明显低于非ED组(19.5±5.2ng/L vs 90±7.5ng/L),两组的PRL、E2、血糖无显著差异,分别为(12.5±2.2μg/L vs 12.7±2.1μg/L)、(36.56±0.53ng/L vs 37.24±0.54ng/L)、(10.2±3.6mmo1/L vs 9.8±3.4mmo1/L)。ED组患者年龄明显高于非ED组(52.4±8.1岁vs 42.4±5.4岁)。两组患者糖尿病病程分别为33.4±10.6月vs 10.9±4.1月,两组相比有显著差异。结论:糖尿病致睾酮的下降是糖尿病性ED产生的原因之一,患者的年龄及糖尿病病程也与糖尿病性ED相关。
文摘In this article, we have reviewed the hepatic perfusion disorder （HPD）, etiopathogenesis of HPD and corresponding diseases. Review of the literature was based on computer searches （PubMed, Index Medicus） and personal experiences. We considered HPD reflects perfusion differences due to redistribution of arterial blood flow among segments, subsegments and lobes of the liver. The plain CT scan findings of HPD manifests as triangular or wedge-shaped areas of low attenuation. On contrast-enhanced CT scan, HPD manifests multiple （or single） transient wedge-shaped, rotundloid or irregular appearance, homogeneous hyperattenuation （in less cases, hypoattenuation） during the hepatic arterial phase （HAP） and isoattenuated or slightly hyperattenuated areas during the portal arterial phase. Dynamic enhanced magnetic resonance （MR） features are similar to enhanced CT scan. Angiographic findings include non-opacification of portal vein on portograms or wedge-shaped segmental staining in arterial and parenchymal phases on hepatic angiograms. The causes of HPD are arterioportal shunts （APS）, intrahepatic vascular compressions and portal vein occlusion, steal phenomenon by hypervascular tumors, vascular variations and any other unknown reasons. It is very important for radiologists to be familiar with the various appearances of HPD to avoid false-positive diagnosis of pseudolesions and not to overestimate the extent of the disease.
