The solid-solid state coord ination reaction enthalpy of CaO w ith ascorb ic ac id has been determ ined by solution calorim e-try in an isoperibel reaction calorim eter at 298.2K.The calorim ietric solvent is 2mol/L H...The solid-solid state coord ination reaction enthalpy of CaO w ith ascorb ic ac id has been determ ined by solution calorim e-try in an isoperibel reaction calorim eter at 298.2K.The calorim ietric solvent is 2mol/L HC l.A thermochem ical cyc le has been de-signed,and the standard Enthalpy of form ation for Ca(C6H7O6)2.4H2O(s) has been calcu lated.The recomm ended values of standard enthalpy of form ationΔfHΘm[Ca(C6H7O6)2.4H2O,s] is-3941.5kJ.展开更多
To improve the reliable performance of information transmission in cooperative relay networks, the scheme of the max-rate spatial channel pairing （SCP） based on maximum ratio combining （MRC） is proposed. The schem...To improve the reliable performance of information transmission in cooperative relay networks, the scheme of the max-rate spatial channel pairing （SCP） based on maximum ratio combining （MRC） is proposed. The scheme includes three steps： channel phase cancellation, MRC, and SCP. Eventually, the solution of the scheme is modeled as convex optimization. The objective function of the optimization problem is to maximize the transmission rate and the optimization variable is the strategy of pairing between the uplink spatial sub-channels of each user and the corresponding downlink spatial ones. The theorem of the arrangement inequalities is adopted to obtain the approximate closed-form solution of the optimal pairing for this convex optimization. Simulation results demonstrate that compared to the existing distributed space-time block coding and coherent combined schemes without SCP, the proposed max-rate SCP plus MRC algorithm achieves appreciable improvements in symbol error rate in medium and high signal-to-noise ratio regimes. The achievable performance gain is due to the use of maxrate SCP.展开更多
AIM： To detect the expression of a proliferation-related ligand on human hepatocellular carcinoma （HCC） cell lines （SK-Hepl, HLE and HepG2） and in culture medium. METHODS： APRIL expression was analyzed by Wester...AIM： To detect the expression of a proliferation-related ligand on human hepatocellular carcinoma （HCC） cell lines （SK-Hepl, HLE and HepG2） and in culture medium. METHODS： APRIL expression was analyzed by Western blotting in HCC cell lines. Effects of APRIL to cell count and angiogenesis were analyzed, too. RESULTS： Recombinant human APRIL （rhAPRIL） increased cell viability of HepG2 cells and, in HUVEC, rhAPRIL provided slight tolerance to cell death from serum starvation. Soluble APRIL （sAPRIL） from HLE cells increased after serum starvation, but did not change in SK-Hepl or HepG2 cells. These cells showed down-regulation of VEGF after incubation with anti-APRIL antibody. Furthermore, culture medium from the HCC cells treated with anti-APRIL antibody treatment inhibited tube formation of HUVECs. CONCLUSION： Functional expression of APRIL might contribute to neovascularization via an upregulation of VEGF in HCC.展开更多
AIM： To characterize the expression and genomic amplification of decoy receptor 3 （DcR3） in hepatocellular carcinoma （HCC） and to evaluate the role of DcR3 in apoptosis.METHODS： We examined 48 cases of HCC for D...AIM： To characterize the expression and genomic amplification of decoy receptor 3 （DcR3） in hepatocellular carcinoma （HCC） and to evaluate the role of DcR3 in apoptosis.METHODS： We examined 48 cases of HCC for DcR3 expression by RT-PCR and DcR3 gene amplification by quantitative genomic PCR. DcR3 protein was detected by immunohistochemistry. Terminal deoxynucleotidyl transferase-mediated dUTP digoxigenin nick and labeling （TUNEL） was used to identify the apoptosis cells in tissues. Primary hepatoma cell culture and MTT test were used to evaluate the protection against FasL- and chemicalinduced apoptosis by DcR3 expression. RESULTS： DcR3 mRNA overexpression was detected in 60% HCC （29/48） patients. The occurrence of HCC was not associated with amplification of the gene. One sample base substitution was found in three sites as a sequence in Genbank. The expression of DcR3 in HCC was associated with the apoptotic index （0.067±0.04 vs 0.209±0.12, P〈0. 01）, size of mass, stage, and infiltration or metastasis （41.2% vs71.0%, 40% vs75%, 51.8% vs84.6%, P〈0. 05）. DcR3 expression could protect hepatoma cells against apoptosis induced by FasL, but not by chemicals. CONCLUSION： These data suggest that in addition to gene amplification there may be another mechanism underlying DcR3 overexpression. The effect of overexpression of DcR3 on the apoptosis of cancer cells may have direct therapeutic implications for the management of HCC.展开更多
文摘The solid-solid state coord ination reaction enthalpy of CaO w ith ascorb ic ac id has been determ ined by solution calorim e-try in an isoperibel reaction calorim eter at 298.2K.The calorim ietric solvent is 2mol/L HC l.A thermochem ical cyc le has been de-signed,and the standard Enthalpy of form ation for Ca(C6H7O6)2.4H2O(s) has been calcu lated.The recomm ended values of standard enthalpy of form ationΔfHΘm[Ca(C6H7O6)2.4H2O,s] is-3941.5kJ.
基金The Open Research Fund of National Mobile Communications Research Laboratory of Southeast University(No.2013D02)the Open Research Fund of National Key Laboratory of Electromagnetic Environment of China Research Institute of Radio Wave Propagation(No.201500013)the National Natural Science Foundation of China(No.61271230,61472190)
文摘To improve the reliable performance of information transmission in cooperative relay networks, the scheme of the max-rate spatial channel pairing （SCP） based on maximum ratio combining （MRC） is proposed. The scheme includes three steps： channel phase cancellation, MRC, and SCP. Eventually, the solution of the scheme is modeled as convex optimization. The objective function of the optimization problem is to maximize the transmission rate and the optimization variable is the strategy of pairing between the uplink spatial sub-channels of each user and the corresponding downlink spatial ones. The theorem of the arrangement inequalities is adopted to obtain the approximate closed-form solution of the optimal pairing for this convex optimization. Simulation results demonstrate that compared to the existing distributed space-time block coding and coherent combined schemes without SCP, the proposed max-rate SCP plus MRC algorithm achieves appreciable improvements in symbol error rate in medium and high signal-to-noise ratio regimes. The achievable performance gain is due to the use of maxrate SCP.
文摘AIM： To detect the expression of a proliferation-related ligand on human hepatocellular carcinoma （HCC） cell lines （SK-Hepl, HLE and HepG2） and in culture medium. METHODS： APRIL expression was analyzed by Western blotting in HCC cell lines. Effects of APRIL to cell count and angiogenesis were analyzed, too. RESULTS： Recombinant human APRIL （rhAPRIL） increased cell viability of HepG2 cells and, in HUVEC, rhAPRIL provided slight tolerance to cell death from serum starvation. Soluble APRIL （sAPRIL） from HLE cells increased after serum starvation, but did not change in SK-Hepl or HepG2 cells. These cells showed down-regulation of VEGF after incubation with anti-APRIL antibody. Furthermore, culture medium from the HCC cells treated with anti-APRIL antibody treatment inhibited tube formation of HUVECs. CONCLUSION： Functional expression of APRIL might contribute to neovascularization via an upregulation of VEGF in HCC.
文摘AIM： To characterize the expression and genomic amplification of decoy receptor 3 （DcR3） in hepatocellular carcinoma （HCC） and to evaluate the role of DcR3 in apoptosis.METHODS： We examined 48 cases of HCC for DcR3 expression by RT-PCR and DcR3 gene amplification by quantitative genomic PCR. DcR3 protein was detected by immunohistochemistry. Terminal deoxynucleotidyl transferase-mediated dUTP digoxigenin nick and labeling （TUNEL） was used to identify the apoptosis cells in tissues. Primary hepatoma cell culture and MTT test were used to evaluate the protection against FasL- and chemicalinduced apoptosis by DcR3 expression. RESULTS： DcR3 mRNA overexpression was detected in 60% HCC （29/48） patients. The occurrence of HCC was not associated with amplification of the gene. One sample base substitution was found in three sites as a sequence in Genbank. The expression of DcR3 in HCC was associated with the apoptotic index （0.067±0.04 vs 0.209±0.12, P〈0. 01）, size of mass, stage, and infiltration or metastasis （41.2% vs71.0%, 40% vs75%, 51.8% vs84.6%, P〈0. 05）. DcR3 expression could protect hepatoma cells against apoptosis induced by FasL, but not by chemicals. CONCLUSION： These data suggest that in addition to gene amplification there may be another mechanism underlying DcR3 overexpression. The effect of overexpression of DcR3 on the apoptosis of cancer cells may have direct therapeutic implications for the management of HCC.
