手足口病（hand，foot and mouth disease．HFMD）是近年来较常见的一种儿童传染性疾病，多发生于春夏秋季，每年都在全国一定范围内暴发流行。手足口病由多种肠道病毒感染所致，以发热、手、足、口腔、臀部出现皮疹为主要特征，少数病...手足口病（hand，foot and mouth disease．HFMD）是近年来较常见的一种儿童传染性疾病，多发生于春夏秋季，每年都在全国一定范围内暴发流行。手足口病由多种肠道病毒感染所致，以发热、手、足、口腔、臀部出现皮疹为主要特征，少数病例可并发神经性肺水肿，无菌性脑膜炎、脑炎、急性弛缓性麻痹和循环衰竭等，展开更多
Objective: To review the pathophysiology and study the diagnosis and clinical management of neurogenic pulmonary edema (NPE). Methods: The data of 5 patients who developed NPE after head injury treated in our hospital...Objective: To review the pathophysiology and study the diagnosis and clinical management of neurogenic pulmonary edema (NPE). Methods: The data of 5 patients who developed NPE after head injury treated in our hospital form December 1995 to May 2003 were collected and analyzed. Results: The patients developed dyspnea and respiratory failure 2-8 hours after neurologic event. Four of the 5 patients presented with pink frothy sputum. Chest radiography showed bilateral diffuse infiltrations in all the 5 patients. After supportive measures such as oxygen support and pharmacologic therapy, 4 patients recovered in 72 hours and one patient died. Conclusions: The pathophysiologic mechanisms of NPE is unclear. In acute respiratory failure following head injury, NPE must be given much attention and timely and effective measures should be taken.展开更多
文摘手足口病（hand，foot and mouth disease．HFMD）是近年来较常见的一种儿童传染性疾病，多发生于春夏秋季，每年都在全国一定范围内暴发流行。手足口病由多种肠道病毒感染所致，以发热、手、足、口腔、臀部出现皮疹为主要特征，少数病例可并发神经性肺水肿，无菌性脑膜炎、脑炎、急性弛缓性麻痹和循环衰竭等，
文摘Objective: To review the pathophysiology and study the diagnosis and clinical management of neurogenic pulmonary edema (NPE). Methods: The data of 5 patients who developed NPE after head injury treated in our hospital form December 1995 to May 2003 were collected and analyzed. Results: The patients developed dyspnea and respiratory failure 2-8 hours after neurologic event. Four of the 5 patients presented with pink frothy sputum. Chest radiography showed bilateral diffuse infiltrations in all the 5 patients. After supportive measures such as oxygen support and pharmacologic therapy, 4 patients recovered in 72 hours and one patient died. Conclusions: The pathophysiologic mechanisms of NPE is unclear. In acute respiratory failure following head injury, NPE must be given much attention and timely and effective measures should be taken.
