Objective: We report a clinical description of pure sensory stroke based on da ta collected from a prospective acute stroke registry. Methods: From 2500 acute stroke patients includedin a hospital-based prospective st...Objective: We report a clinical description of pure sensory stroke based on da ta collected from a prospective acute stroke registry. Methods: From 2500 acute stroke patients includedin a hospital-based prospective stroke registry over a 12-year period, 99 were identified as having pure sensory stroke. Results:Pure sensory stroke accounted for 4.7%of all acute stroke patients, 5.4%of acute is chemic stroke, and 17.4%of lacunar syndromes. Complete hemisensory syndrome was present in 80 patients and incomplete hemisensory syndrome in 19 (cheirooral sy ndrome 12, cheiro-oral-pedal 6, isolated oral syndrome1). The lacunar hypothes is was fulfilled in 88%of patients.Atherothrombotic infarction occurred in 8 pa tients, intracerebral hemorrhage in 3, and stroke of undetermined cause in 1.Hem orrhagic pure sensory stroke was diagnosed in 1%of all cases of hemorrhagic str oke (n=270) in the database. Outcome was good (in-hospital mortality 0%, sympt om-free at discharge 41.5 %). After multivariate analysis, absence of disabili ty at discharge, hypertension, diabetes, hyperlipidemia, and thalamic(56.5 %) a nd corona radiata (4 %) locations were clinical and topographic variables signi ficantly associated with pure sensory stroke. Conclusions: Pure sensory stroke i s an infrequent cerebrovascular syndrome, in which the lacunar hypothesis is sup ported. Most patients had thalamic lacunar infarction. Incomplete hemisensory sy ndromes were also caused by a lacunar infarct in 84%of patients. Hemorrhagic pu re sensory stroke accounted only for 3%of the cases. The prognosis is good with striking similarity to other lacunar strokes. There are important differences b etween pure sensory stroke and nonlacunar strokes.展开更多
文摘Objective: We report a clinical description of pure sensory stroke based on da ta collected from a prospective acute stroke registry. Methods: From 2500 acute stroke patients includedin a hospital-based prospective stroke registry over a 12-year period, 99 were identified as having pure sensory stroke. Results:Pure sensory stroke accounted for 4.7%of all acute stroke patients, 5.4%of acute is chemic stroke, and 17.4%of lacunar syndromes. Complete hemisensory syndrome was present in 80 patients and incomplete hemisensory syndrome in 19 (cheirooral sy ndrome 12, cheiro-oral-pedal 6, isolated oral syndrome1). The lacunar hypothes is was fulfilled in 88%of patients.Atherothrombotic infarction occurred in 8 pa tients, intracerebral hemorrhage in 3, and stroke of undetermined cause in 1.Hem orrhagic pure sensory stroke was diagnosed in 1%of all cases of hemorrhagic str oke (n=270) in the database. Outcome was good (in-hospital mortality 0%, sympt om-free at discharge 41.5 %). After multivariate analysis, absence of disabili ty at discharge, hypertension, diabetes, hyperlipidemia, and thalamic(56.5 %) a nd corona radiata (4 %) locations were clinical and topographic variables signi ficantly associated with pure sensory stroke. Conclusions: Pure sensory stroke i s an infrequent cerebrovascular syndrome, in which the lacunar hypothesis is sup ported. Most patients had thalamic lacunar infarction. Incomplete hemisensory sy ndromes were also caused by a lacunar infarct in 84%of patients. Hemorrhagic pu re sensory stroke accounted only for 3%of the cases. The prognosis is good with striking similarity to other lacunar strokes. There are important differences b etween pure sensory stroke and nonlacunar strokes.
