目的探讨鼓膜穿孔部位、面积及钙化斑对患者听力的影响。方法选择慢性化脓性中耳炎静止期患者71例,检测患者听阈,分别记录0. 5、1、2、4 k Hz四个频率的气导听阈(AC)、骨导听阈(BC)、气骨导差(ABG);用耳内镜检测穿孔位置、面积、钙化斑...目的探讨鼓膜穿孔部位、面积及钙化斑对患者听力的影响。方法选择慢性化脓性中耳炎静止期患者71例,检测患者听阈,分别记录0. 5、1、2、4 k Hz四个频率的气导听阈(AC)、骨导听阈(BC)、气骨导差(ABG);用耳内镜检测穿孔位置、面积、钙化斑。分析鼓膜穿孔部位、面积、钙化斑对听阈的影响。结果不同穿孔部位的AC、ABG均为0. 5 k Hz时阈值最高,2 k Hz时最低,4 k Hz时升高;相同频率,鼓膜后下方穿孔阈值高于前下方,大穿孔AC、ABG阈值最高;但不同部位穿孔的各频率AC、BC、ABG阈值比较差异均无统计学意义(P均> 0. 05)。穿孔面积1度与4度、2度与4度穿孔时各频率AC阈值均不同,3度与4度除2 k Hz外,其余各频率AC阈值比较差异有统计学意义(P均<0. 05)。不同穿孔面积各频率BC比较差异无统计学意义(P均> 0. 05);不同穿孔面积在0. 5、1 k Hz时ABG比较差异有统计学意义(P均<0. 05)。有鼓膜钙化斑与无鼓膜钙化斑患者各频率AC、BC、ABG比较差异无统计学意义(P均> 0. 05)。结论鼓膜穿孔面积大的患者听力下降明显,鼓膜穿孔部位、钙化斑对患者听力无明显影响。展开更多
Electron beam computed tomographic studies have demonstrated that the extent of intracoronary calcium is related to risk of coronary events. This study was performed to gain further insight into the distribution of fo...Electron beam computed tomographic studies have demonstrated that the extent of intracoronary calcium is related to risk of coronary events. This study was performed to gain further insight into the distribution of focal calcifications and their relation to the site of plaque rupture within the culprit artery in consecutive patients(n=60) with acute myocardial infarction(AMI) using intravascular ultrasound imaging. Calcifications in the culprit lesion and adjacent segments were classified and counted according to their arc(< 45°, 45°to 90°, 90°to 180°, >180°), length(< 1.5, 1.5 to 3.0, 3.0 to 6.0, >6.0 mm), and dispersion(number of spots per millimeter). Calcifications at the edge of a visible rupture or ulceration were considered related to the AMI. Compared with adjacent proximal and distal segments, the culprit lesion contained more calcified spots per millimeter(0.14, 0.10, and 0.21, respectively, p< 0.05). Small calcified spots(arc< 45°, length< 1.5 mm) were more common(p< 0.05). Plaque rupture or ulceration was manifest in 31 culprit lesions(52%), 14(45%) of which contained focal calcifications. These calcified spots extended more often to 90°to 180°of the vessel circumference and were more often of moderate length(3 to 6 mm) compared with culprit lesions without visible plaque rupture(p< 0.05). In conclusion, culprit lesions in patients with AMI contain more and smaller calcifications compared with adjacent segments. Calcifications related to plaque rupture appear to be larger and extend over a wider arc compared with these calcified spots. Those larger calcified spots may play a role in plaque instability in a subgroup of lesions.展开更多
文摘目的探讨鼓膜穿孔部位、面积及钙化斑对患者听力的影响。方法选择慢性化脓性中耳炎静止期患者71例,检测患者听阈,分别记录0. 5、1、2、4 k Hz四个频率的气导听阈(AC)、骨导听阈(BC)、气骨导差(ABG);用耳内镜检测穿孔位置、面积、钙化斑。分析鼓膜穿孔部位、面积、钙化斑对听阈的影响。结果不同穿孔部位的AC、ABG均为0. 5 k Hz时阈值最高,2 k Hz时最低,4 k Hz时升高;相同频率,鼓膜后下方穿孔阈值高于前下方,大穿孔AC、ABG阈值最高;但不同部位穿孔的各频率AC、BC、ABG阈值比较差异均无统计学意义(P均> 0. 05)。穿孔面积1度与4度、2度与4度穿孔时各频率AC阈值均不同,3度与4度除2 k Hz外,其余各频率AC阈值比较差异有统计学意义(P均<0. 05)。不同穿孔面积各频率BC比较差异无统计学意义(P均> 0. 05);不同穿孔面积在0. 5、1 k Hz时ABG比较差异有统计学意义(P均<0. 05)。有鼓膜钙化斑与无鼓膜钙化斑患者各频率AC、BC、ABG比较差异无统计学意义(P均> 0. 05)。结论鼓膜穿孔面积大的患者听力下降明显,鼓膜穿孔部位、钙化斑对患者听力无明显影响。
文摘Electron beam computed tomographic studies have demonstrated that the extent of intracoronary calcium is related to risk of coronary events. This study was performed to gain further insight into the distribution of focal calcifications and their relation to the site of plaque rupture within the culprit artery in consecutive patients(n=60) with acute myocardial infarction(AMI) using intravascular ultrasound imaging. Calcifications in the culprit lesion and adjacent segments were classified and counted according to their arc(< 45°, 45°to 90°, 90°to 180°, >180°), length(< 1.5, 1.5 to 3.0, 3.0 to 6.0, >6.0 mm), and dispersion(number of spots per millimeter). Calcifications at the edge of a visible rupture or ulceration were considered related to the AMI. Compared with adjacent proximal and distal segments, the culprit lesion contained more calcified spots per millimeter(0.14, 0.10, and 0.21, respectively, p< 0.05). Small calcified spots(arc< 45°, length< 1.5 mm) were more common(p< 0.05). Plaque rupture or ulceration was manifest in 31 culprit lesions(52%), 14(45%) of which contained focal calcifications. These calcified spots extended more often to 90°to 180°of the vessel circumference and were more often of moderate length(3 to 6 mm) compared with culprit lesions without visible plaque rupture(p< 0.05). In conclusion, culprit lesions in patients with AMI contain more and smaller calcifications compared with adjacent segments. Calcifications related to plaque rupture appear to be larger and extend over a wider arc compared with these calcified spots. Those larger calcified spots may play a role in plaque instability in a subgroup of lesions.
