We investigate the Luminosity Function （LF） of the cluster of galaxies Abell 566. The photometric data of 15 intermediate-bands are obtained from the Beijing-Arizona- Taiwan-Connecticut （BATC） photometric sky surv...We investigate the Luminosity Function （LF） of the cluster of galaxies Abell 566. The photometric data of 15 intermediate-bands are obtained from the Beijing-Arizona- Taiwan-Connecticut （BATC） photometric sky survey. For each of the 15 wavebands, the LF of cluster galaxies is well modelled by the Schechter function, with characteristic luminosities from -18.0 to -21.9 magnitude, from the α- to the p-band. Morphological dependence of the LF is investigated by separating the cluster members into ＇red＇ and ＇blue＇ subsamples. It is clear that late type galaxies have a steeper shape of LF than the early type galaxies. We also divided the sample galaxies by their local environment. It was found that galaxies in the sparser region have steeper shape of LF than galaxies in the denser region. Combining the results of morphological and environmental dependence of LFs, we show that Abell 566 is a well relaxed cluster with positive evidence of galaxy interaction and merger, and excess number of bright early type galaxies located in its denser region.展开更多
It has been certified that GABPB1-AS1 is aberrantly expressed and plays as a vital role in some kinds of cancers.However,its expression pattern and functions in non-small cell lung cancer(NSCLC)are still largely unknow...It has been certified that GABPB1-AS1 is aberrantly expressed and plays as a vital role in some kinds of cancers.However,its expression pattern and functions in non-small cell lung cancer(NSCLC)are still largely unknown.This study aims to assess GABPB1-AS1 expression and biological roles in NSCLC.The expression of GABPB1-AS1 was detected in NSCLC specimens and adjacent normal specimens.CCK8 and Transwell assays were performed to evaluate the effects of GABPB1-AS1 on NSCLC cell proliferation,migration and invasion.Bioinformatics tools and luciferase reporter assays were applied to predict and verify GABPB1-AS1’s direct targets.The results revealed that GABPB1-AS1 is sharply reduced in NSCLC specimens and cell lines.CCK8 assays indicated that overexpression of GABPB1-AS1 dramatically reduced NSCLC cell growth,and Transwell assays proved that NSCLC cell migration and invasion were distinctly inhibited by GABPB1-AS1.Exploration of the mechanism uncovered that miRNA-566(miR-566)/F-box protein 47(FBXO47)is directly targeted by GABPB1-AS1 in NSCLC.The study demonstrated that GABPB1-AS1 inhibited NSCLC cell proliferation,migration and invasion by targeting miR-566/FBXO47.展开更多
作为全球知名的板卡厂商，微星早已被大家所熟知，在随身数码设备逐渐走红的今天，面对新兴的“随身看”市场，微星适宜时机地先后推出了两款“随身看”产品，MSI MEGA VIEW 566（以下简称VIEW 566）就是其旗舰产品。从之前的MP3产品精...作为全球知名的板卡厂商，微星早已被大家所熟知，在随身数码设备逐渐走红的今天，面对新兴的“随身看”市场，微星适宜时机地先后推出了两款“随身看”产品，MSI MEGA VIEW 566（以下简称VIEW 566）就是其旗舰产品。从之前的MP3产品精致的做工就可以看出，微星对产品做工的一丝不苟。展开更多
基金Supported by the National Natural Science Foundation of China.
文摘We investigate the Luminosity Function （LF） of the cluster of galaxies Abell 566. The photometric data of 15 intermediate-bands are obtained from the Beijing-Arizona- Taiwan-Connecticut （BATC） photometric sky survey. For each of the 15 wavebands, the LF of cluster galaxies is well modelled by the Schechter function, with characteristic luminosities from -18.0 to -21.9 magnitude, from the α- to the p-band. Morphological dependence of the LF is investigated by separating the cluster members into ＇red＇ and ＇blue＇ subsamples. It is clear that late type galaxies have a steeper shape of LF than the early type galaxies. We also divided the sample galaxies by their local environment. It was found that galaxies in the sparser region have steeper shape of LF than galaxies in the denser region. Combining the results of morphological and environmental dependence of LFs, we show that Abell 566 is a well relaxed cluster with positive evidence of galaxy interaction and merger, and excess number of bright early type galaxies located in its denser region.
基金supported by the Highlevel Hospital Construction Research Project of Maoming People’s Hospital[Grant No.zx2020012]Maoming Science and Technology Project Special Fund in 2020[Grant No.2020KJZX014]Maoming Science and Technology Project[Grant No.2020395].
文摘It has been certified that GABPB1-AS1 is aberrantly expressed and plays as a vital role in some kinds of cancers.However,its expression pattern and functions in non-small cell lung cancer(NSCLC)are still largely unknown.This study aims to assess GABPB1-AS1 expression and biological roles in NSCLC.The expression of GABPB1-AS1 was detected in NSCLC specimens and adjacent normal specimens.CCK8 and Transwell assays were performed to evaluate the effects of GABPB1-AS1 on NSCLC cell proliferation,migration and invasion.Bioinformatics tools and luciferase reporter assays were applied to predict and verify GABPB1-AS1’s direct targets.The results revealed that GABPB1-AS1 is sharply reduced in NSCLC specimens and cell lines.CCK8 assays indicated that overexpression of GABPB1-AS1 dramatically reduced NSCLC cell growth,and Transwell assays proved that NSCLC cell migration and invasion were distinctly inhibited by GABPB1-AS1.Exploration of the mechanism uncovered that miRNA-566(miR-566)/F-box protein 47(FBXO47)is directly targeted by GABPB1-AS1 in NSCLC.The study demonstrated that GABPB1-AS1 inhibited NSCLC cell proliferation,migration and invasion by targeting miR-566/FBXO47.
