In this paper we present a regularized Newton-type method for ill-posed problems, by using the A-smooth regularization to solve the linearized ill-posed equations. For noisy data a proper a posteriori stopping rule is...In this paper we present a regularized Newton-type method for ill-posed problems, by using the A-smooth regularization to solve the linearized ill-posed equations. For noisy data a proper a posteriori stopping rule is used that yields convergence of the Newton iteration to a solution, as the noise level goes to zero, under certain smoothness conditions on the nonlinear operator. Some appropriate assumptions on the closedness and smoothness of the starting value and the solution are shown to lead to optimal convergence rates.展开更多
In the present study, retinal MOiler cells were cultured in vitro and treated with hydrogen peroxide （oxidative stressor） and cobalt chloride （hypoxic injury）. Following 24 hours of culture, compensatory hypertrop...In the present study, retinal MOiler cells were cultured in vitro and treated with hydrogen peroxide （oxidative stressor） and cobalt chloride （hypoxic injury）. Following 24 hours of culture, compensatory hypertrophy was observed and cellular apoptosis increased. Hypoxia enhanced the migration ability of retinal MOiler cells and induced the expression of a-smooth muscle actin. Oxidative stress altered the morphology of MOiler cells when compared with hypoxia treatment.展开更多
文摘In this paper we present a regularized Newton-type method for ill-posed problems, by using the A-smooth regularization to solve the linearized ill-posed equations. For noisy data a proper a posteriori stopping rule is used that yields convergence of the Newton iteration to a solution, as the noise level goes to zero, under certain smoothness conditions on the nonlinear operator. Some appropriate assumptions on the closedness and smoothness of the starting value and the solution are shown to lead to optimal convergence rates.
文摘In the present study, retinal MOiler cells were cultured in vitro and treated with hydrogen peroxide （oxidative stressor） and cobalt chloride （hypoxic injury）. Following 24 hours of culture, compensatory hypertrophy was observed and cellular apoptosis increased. Hypoxia enhanced the migration ability of retinal MOiler cells and induced the expression of a-smooth muscle actin. Oxidative stress altered the morphology of MOiler cells when compared with hypoxia treatment.
