Postoperative pleural effusion occurs frequently after hepatectomy. The risk factors, prevention and management of postoperative pleural effusion in patients with primary liver cancer (PLC) who have undergone hepatect...Postoperative pleural effusion occurs frequently after hepatectomy. The risk factors, prevention and management of postoperative pleural effusion in patients with primary liver cancer (PLC) who have undergone hepatectomy and the value of the argon beam coagulator (ABC) for the prevention of pleural effusion are studied. METHODS:A total of 523 patients with PLC at our institution who had had right hepatectomy from July 2000 to June 2004 were studied retrospectively. Comparative analysis was made to identify the factors contributing to postoperative pleural effusion and the efficacy of various managements. RESULTS:Of the 523 patients whose livers were dissociated using argon beam cutting and/or coagulation, 20(3.8%) developed pleural effusions;whereas in the other 467 patients underwent hepatectomy with suture ligation of the diaphragmatic secondary wound surface during the same period, 49(10.5%) had pleural effusion (P<0.01). The factors contributing to postoperative pleural effusion included subphrenic collection, postoperative hepatic insufficiency with ascites, duration of hepatic occlusion and underlying cirrhosis. CONCLUSIONS: Dissociation of the liver by argon beam cutting and/or coagulation can save suture ligation of the diaphragmatic secondary wound surface and may also prevent postoperative pleural effusion. Pleural drainage using an indwelling central-venous-catheter (CVC) in the pleural cavity is safe and efficacious.展开更多
目的探讨经外周中心静脉置管(peripherally inserted central catheters,PICC)引发的新生儿严重并发症的发病特点、临床和影像学表现,以及治疗转归情况。方法回顾性分析2015年1月至2020年5月中国医科大学附属盛京医院新生儿科收治、因P...目的探讨经外周中心静脉置管(peripherally inserted central catheters,PICC)引发的新生儿严重并发症的发病特点、临床和影像学表现,以及治疗转归情况。方法回顾性分析2015年1月至2020年5月中国医科大学附属盛京医院新生儿科收治、因PICC置管引发胸腔积液、心律失常及心包积液患儿的临床资料,总结发病特点、临床和影像学表现,以及治疗转归情况。结果共纳入9例患儿,均为极低或超低出生体重儿,均存活。其中8例PICC导管异位,1例导管位置正常(心律失常患儿)。6例胸腔积液患儿中双侧1例,右侧5例,均在原有呼吸支持下出现呼吸困难、频繁呼吸暂停,需气管插管、机械通气,胸水特点均为葡萄糖明显增高,细胞数及蛋白无明显增高,予胸腔穿刺并拔出PICC导管后症状很快好转,均未再出现胸腔积液。2例心律失常患儿分别表现为心房扑动伴室性心动过速及室上性心动过速,应用抗心律失常药物后无改善,拔出PICC后2~12 h内复律,且未再出现心律失常。1例心包积液患儿出现心脏增大、心音遥远及心率下降等心包填塞症状,予心包穿刺后上述症状很快消失,积液生化特点同PICC引发胸腔积液的胸水生化特点。结论PICC可引发新生儿严重甚至致死性并发症,当患儿出现不明原因胸腔积液、心律失常、心包积液甚至心包填塞,或不明原因病情变化无法用常规现象解释时,需警惕是否为PICC引发,危重病例治疗并发症同时应尽早拔出PICC导管。展开更多
文摘Postoperative pleural effusion occurs frequently after hepatectomy. The risk factors, prevention and management of postoperative pleural effusion in patients with primary liver cancer (PLC) who have undergone hepatectomy and the value of the argon beam coagulator (ABC) for the prevention of pleural effusion are studied. METHODS:A total of 523 patients with PLC at our institution who had had right hepatectomy from July 2000 to June 2004 were studied retrospectively. Comparative analysis was made to identify the factors contributing to postoperative pleural effusion and the efficacy of various managements. RESULTS:Of the 523 patients whose livers were dissociated using argon beam cutting and/or coagulation, 20(3.8%) developed pleural effusions;whereas in the other 467 patients underwent hepatectomy with suture ligation of the diaphragmatic secondary wound surface during the same period, 49(10.5%) had pleural effusion (P<0.01). The factors contributing to postoperative pleural effusion included subphrenic collection, postoperative hepatic insufficiency with ascites, duration of hepatic occlusion and underlying cirrhosis. CONCLUSIONS: Dissociation of the liver by argon beam cutting and/or coagulation can save suture ligation of the diaphragmatic secondary wound surface and may also prevent postoperative pleural effusion. Pleural drainage using an indwelling central-venous-catheter (CVC) in the pleural cavity is safe and efficacious.
文摘目的探讨经外周中心静脉置管(peripherally inserted central catheters,PICC)引发的新生儿严重并发症的发病特点、临床和影像学表现,以及治疗转归情况。方法回顾性分析2015年1月至2020年5月中国医科大学附属盛京医院新生儿科收治、因PICC置管引发胸腔积液、心律失常及心包积液患儿的临床资料,总结发病特点、临床和影像学表现,以及治疗转归情况。结果共纳入9例患儿,均为极低或超低出生体重儿,均存活。其中8例PICC导管异位,1例导管位置正常(心律失常患儿)。6例胸腔积液患儿中双侧1例,右侧5例,均在原有呼吸支持下出现呼吸困难、频繁呼吸暂停,需气管插管、机械通气,胸水特点均为葡萄糖明显增高,细胞数及蛋白无明显增高,予胸腔穿刺并拔出PICC导管后症状很快好转,均未再出现胸腔积液。2例心律失常患儿分别表现为心房扑动伴室性心动过速及室上性心动过速,应用抗心律失常药物后无改善,拔出PICC后2~12 h内复律,且未再出现心律失常。1例心包积液患儿出现心脏增大、心音遥远及心率下降等心包填塞症状,予心包穿刺后上述症状很快消失,积液生化特点同PICC引发胸腔积液的胸水生化特点。结论PICC可引发新生儿严重甚至致死性并发症,当患儿出现不明原因胸腔积液、心律失常、心包积液甚至心包填塞,或不明原因病情变化无法用常规现象解释时,需警惕是否为PICC引发,危重病例治疗并发症同时应尽早拔出PICC导管。