The effect of oral L-ornithine hydrochloride (0.1 g/kg BW) on energy expenditure during a rest period from 120 to 180 min after resistance exercise was evaluated by indirect calorimetry. Healthy male subjects who have...The effect of oral L-ornithine hydrochloride (0.1 g/kg BW) on energy expenditure during a rest period from 120 to 180 min after resistance exercise was evaluated by indirect calorimetry. Healthy male subjects who have no habit of resistance training underwent resistance exercise (chest press, lat pulldown, leg press, shoulder press, leg extension, and leg curl), with 3 sets of each exercise and 10 repetitions in each set at 90-s intervals, 30 min after ingestion of ornithine or placebo. Plasma ornithine levels immediately after, and 120 and 180 min after, resistance exercise were significantly greater after ingestion of ornithine than of placebo (Treatment: F = 347.1, P p2 = 0.95;Interaction: F = 160.7, P p2 = 0.95), but no significant difference in serum growth hormone levels was observed between the two treatments (Treatment: F = 0.1, P = 0.751, ηp2 = 0.01;Time: F = 1.7, P = 0.229, ηp2 = 0.16;Interaction: F = 2.4, P = 0.155, ηp2 = 0.21). Although there was no between-treatment difference in energy expenditure during the rest period 120 to 180 min after resistance exercise (Treatment: F = 0.1, P = 0.718, ηp2 = 0.02;Time: F = 0.1, P = 0.767, ηp2 = 0.01;Interaction: F = 0.1, P = 0.112, ηp2 = 0.26), with ornithine ingestion carbohydrate oxidation was significantly greater than with placebo from 170 to 180 min after exercise (Treatment: F = 0.8, P = 0.383, ηp2 = 0.09;Time: F = 9.7, P = 0.013, ηp2 = 0.52;Interaction: F = 5.8, P = 0.039, ηp2 = 0.39). Moreover, 180 min after exercise, serum free fatty acid levels after ornithine ingestion were lower than after placebo (Treatment: F = 0.3, P = 0.602, ηp2 = 0.03;Time: F = 34.6, P p2 = 0.79;Interaction: F = 5.6, P = 0.042, ηp2 = 0.38). A similar trend in 3-hydroxybutylate was observed. In conclusion, ornithine ingestion before resistance exercise may enhance post-exercise carbohydrate oxidation without changing total energy expenditure.展开更多
目的:观察认知障碍诊治系统ZM3.1对卒中后认知功能障碍患者的康复疗效。方法:入选50例脑卒中后认知功能障碍患者,采用单盲随机对照方法分为试验组和对照组,每组25例,其中试验组予以认知障碍诊治系统ZM3.1训练系统进行认知功能训练,对照...目的:观察认知障碍诊治系统ZM3.1对卒中后认知功能障碍患者的康复疗效。方法:入选50例脑卒中后认知功能障碍患者,采用单盲随机对照方法分为试验组和对照组,每组25例,其中试验组予以认知障碍诊治系统ZM3.1训练系统进行认知功能训练,对照组采用传统认知功能训练,均治疗4周,治疗前后采用简易智能状态量表(mini-mental state examination,MMSE)评定,比较分析治疗前后患者时间定向力、地点定向力、复述能力、计算能力、记忆能力、辨认能力、理解能力、表达能力、结构模仿能力9项认知功能亚项能力。结果:(1)试验组治疗后总评分高于治疗前,且时间定向力、地点定向力、复述能力、计算能力、记忆能力、理解能力等认知功能亚项评分高于治疗前,差异均具有显著性意义(P<0.05);(2)对照组治疗后总评分高于治疗前,且时间定向力、地点定向力、记忆能力、理解能力等认知功能亚项评分高于治疗前,差异均具有显著性意义(P<0.05);(3)治疗后试验组在时间定向力、计算能力等认知功能亚项的改善优于对照组(P<0.05)。结论:认知障碍诊治系统ZM3.1的训练系统治疗可改善脑卒中后认知功能障碍,与传统认知康复训练相比,在时间定向力和计算能力改善方面具有一定的优势。展开更多
文摘The effect of oral L-ornithine hydrochloride (0.1 g/kg BW) on energy expenditure during a rest period from 120 to 180 min after resistance exercise was evaluated by indirect calorimetry. Healthy male subjects who have no habit of resistance training underwent resistance exercise (chest press, lat pulldown, leg press, shoulder press, leg extension, and leg curl), with 3 sets of each exercise and 10 repetitions in each set at 90-s intervals, 30 min after ingestion of ornithine or placebo. Plasma ornithine levels immediately after, and 120 and 180 min after, resistance exercise were significantly greater after ingestion of ornithine than of placebo (Treatment: F = 347.1, P p2 = 0.95;Interaction: F = 160.7, P p2 = 0.95), but no significant difference in serum growth hormone levels was observed between the two treatments (Treatment: F = 0.1, P = 0.751, ηp2 = 0.01;Time: F = 1.7, P = 0.229, ηp2 = 0.16;Interaction: F = 2.4, P = 0.155, ηp2 = 0.21). Although there was no between-treatment difference in energy expenditure during the rest period 120 to 180 min after resistance exercise (Treatment: F = 0.1, P = 0.718, ηp2 = 0.02;Time: F = 0.1, P = 0.767, ηp2 = 0.01;Interaction: F = 0.1, P = 0.112, ηp2 = 0.26), with ornithine ingestion carbohydrate oxidation was significantly greater than with placebo from 170 to 180 min after exercise (Treatment: F = 0.8, P = 0.383, ηp2 = 0.09;Time: F = 9.7, P = 0.013, ηp2 = 0.52;Interaction: F = 5.8, P = 0.039, ηp2 = 0.39). Moreover, 180 min after exercise, serum free fatty acid levels after ornithine ingestion were lower than after placebo (Treatment: F = 0.3, P = 0.602, ηp2 = 0.03;Time: F = 34.6, P p2 = 0.79;Interaction: F = 5.6, P = 0.042, ηp2 = 0.38). A similar trend in 3-hydroxybutylate was observed. In conclusion, ornithine ingestion before resistance exercise may enhance post-exercise carbohydrate oxidation without changing total energy expenditure.
文摘目的:观察认知障碍诊治系统ZM3.1对卒中后认知功能障碍患者的康复疗效。方法:入选50例脑卒中后认知功能障碍患者,采用单盲随机对照方法分为试验组和对照组,每组25例,其中试验组予以认知障碍诊治系统ZM3.1训练系统进行认知功能训练,对照组采用传统认知功能训练,均治疗4周,治疗前后采用简易智能状态量表(mini-mental state examination,MMSE)评定,比较分析治疗前后患者时间定向力、地点定向力、复述能力、计算能力、记忆能力、辨认能力、理解能力、表达能力、结构模仿能力9项认知功能亚项能力。结果:(1)试验组治疗后总评分高于治疗前,且时间定向力、地点定向力、复述能力、计算能力、记忆能力、理解能力等认知功能亚项评分高于治疗前,差异均具有显著性意义(P<0.05);(2)对照组治疗后总评分高于治疗前,且时间定向力、地点定向力、记忆能力、理解能力等认知功能亚项评分高于治疗前,差异均具有显著性意义(P<0.05);(3)治疗后试验组在时间定向力、计算能力等认知功能亚项的改善优于对照组(P<0.05)。结论:认知障碍诊治系统ZM3.1的训练系统治疗可改善脑卒中后认知功能障碍,与传统认知康复训练相比,在时间定向力和计算能力改善方面具有一定的优势。
