Change of Tropical Cyclone Heat Potential in Response to Global Warming

Tropical cyclone heat potential （TCHP） in the ocean can affect tropical cyclone intensity and intensification. In this paper, TCHP change under global warming is presented based on 35 models from CMIP5 （Coupled Model Intercomparison Project, Phase 5）. As the upper ocean warms up, the TCHP of the global ocean is projected to increase by 140.6% in the 21st century under the RCP4.5 （＋4.5 W m 2 Representative Concentration Pathway） scenario, The increase is particularly significant in the western Pacific, northwestern Indian and western tropical Atlantic oceans. The increase of TCHP results from the ocean temperature warming above the depth of the 26~C isotherm （D26）, the deepening of D26, and the horizontal area expansion of SST above 26~C. Their contributions are 69.4%, 22.5% and 8.1%, respectively. Further, a suite of numerical experiments with an Ocean General Circulation Model （OGCM） is conducted to investigate the relative importance of wind stress and buoyancy forcing to the TCHP change under global warming. Results show that sea surface warming is the dominant forcing for the TCHP change, while wind stress and sea surface salinity change are secondary.

Potential enstrophical diagnostic analyses on the mechanism of change of tropical cyclone intensity

It is indicated that the change of mean potential enstrophy within tropical cyclone (TC) corresponds tothe change of TC intensity. A series of factors influencing the intensity change have been discussed by calculating budget of potential enstrophy in tangential wave-number domain in cylindric coordinates. The results indicate thatthe vertical distribution of mean diabatic heating is an important factor that influences the change of TC intensitythrough transformation mechanism of the Coriolis effect and cyclonic vorticity, especially in the sudden intensifyingstage of TC. It is favourable to the intensification when the diabatic heating is largest in upper-middle troposphere,while TC weakens when this kind of role of heating become small or the heating is largest in lower troposphere. Therole of the axisymmetric fields of TC is different from that of the non-axisymmetric fields. In addition,we have analysed some other factors that influence TC intensity.

Satellite derived upper ocean thermal structure and its application to tropical cyclone intensity forecasting in the Indian Ocean

Upper ocean heat content is a factor critical to the intensity change of tropical cyclones(TCs). Because of the inhomogeneity of in situ observations in the North Indian Ocean,gridded temperature/salinity(T/S) profiles were derived from satellite data for 1993–2012 using a linear regression method. The satellite derived T/S dataset covered the region of 10°S–32°N,25°–100°E with daily temporal resolution,0.25°×0.25° spatial resolution,and 26 vertical layers from the sea surface to a depth of 1 000 m at standard layers. Independent Global Temperature Salinity Profile Project data were used to validate the satellite derived T/S fields. The analysis confirmed that the satellite derived temperature field represented the characteristics and vertical structure of the temperature field well. The results demonstrated that the vertically averaged root mean square error of the temperature was 0.83 in the upper 1 000 m and the corresponding correlation coefficient was 0.87,which accounted for 76% of the observed variance. After verification of the satellite derived T/S dataset,the TC heat potential(TCHP) was verified. The results show that the satellite derived values were coherent with observed TCHP data with a correlation coefficient of 0.86 and statistical significance at the 99% confidence level. The intensity change of TC Gonu during a period of rapid intensification was studied using satellite derived TCHP data. A delayed effect of the TCHP was found in relation to the intensity change of Gonu,suggesting a lag feature in the response of the inner core of the TC to the ocean.

The Pressure Gradient Elastic Wave： Energy Transfer Process for Compressible Fluids with Pressure Gradient

The temperature separation was discovered inside the short vortex chamber （H/D = 0.18）. Experiments revealed that the highest temperature of the periphery was 465 ℃, and the lowest temperature of the central zone was -45 ℃ （the compressed air was pumped into the chamber at room temperature）. The objective of this paper is to proof that this temperature separation effect cannot be explained by conventional heat transfer processes. To explain this phenomenon, the concept of PGEW （Pressure Gradient Elastic Waves） is proposed. PGEW are kind of elastic waves, which operate in compressible fluids with pressure gradients and density fluctuations. The result of PGEW propagation is a heat transfer from area of low pressure to high pressure zone. The physical model of a gas in a strong field of mass forces is proposed to substantiate the PGEW existence. This physical model is intended for the construction of a theory of PGEW. Understanding the processes associated with the PGEW permits the possibility of creating new devices for energy saving and low potential heat utilization, which have unique properties.

Role of transient receptor potential vanilloid subetype 1 in the increase of thermal pain threshold by moxibustion

OBTECTIVE:To explore the role of transient receptor potential vaniiloid subetype 1(TRPV1) in the increase of the thermal pain threshold by moxibustion.METHODS:Forty Kunming mice(20 ± 2) g were randomized into control group,capsaicin group,capsazepine group,moxibustion group and moxibustion + capsazepine(MC) group with 8 mice in each,and 16 C57BL/6 wild-type mice(18 ± 2) g were randomized into wild-type(WT) control group and WT moxibustion group with 8 mice in each,and 14 TRPV1 knockout mice(18 ± 2) g were randomized into knockout(KO) control group and KO moxibustion group with 7 in each.Each mouse in the capsaicin group was subcutaneously injected with the amount of 0.1 mL/10 g into L5 and L6 spinal cords;each mouse in the capsazepine group was intraperitoneally injected with the amount of0.1 mL/10 g.Similarly,each mouse in the moxibustion group was given a suspended moxibustion with specially-made moxa-stick for 20 min on L5 and L6 spinal cords.Each mouse in MC group was intraperitoneally injected with the amount of 0.1 mL/10 g first,then after 15 min was given a suspended moxibustion for 20 min on L5 and L6 spinal cords.Each mouse in WT moxibustion group and KO moxibustion group was given a suspended moxibustion with specially-made moxa-stick for 20 min on L5 and L6 spinal cords.The control group,WT control group and KO control group were of no treatment in any way.After all treatments were completed,the digital-display measurement instrument for thermal pain was used to measure the threshold of thermal pain in each group respectively.RESULTS:Compared with the control group,the thresholds of thermal pain in the moxibustion group and MC group were significantly increased(P <0.01);no significant changes in the thresholds in the capsaicin group and the capsazepine group(P > 0.05);compared with moxibustion group,he threshold of thermal in MC group was obviously decreased(P < 0.01).Compared with WT control group,the threshold of thermal pain in WT moxibustion group was significantly increased(P <0.01);compared with KO control group,no changes in the threshold in KO moxibustion group(P > 0.05).CONCLUSION:TRPV1 participated in the process of increasing the threshold of thermal pain by stimulating L5 and L6 of mice spinal cord with burning mosa-stick.